G6PD Deficiency Food To Avoid Some of the foods commonly eaten around the world can cause people with G6PD Deficiency to hemolyze hemolyze.. Some of these foods can be deadly (like fava beans). Some others can cause low level hemolysis which means that red blood cells die but not enou!h to cause the person to !o to the hospital. "ow level hemolysis over time can cause other problems such as memory dysfunction overworked spleen liver kidney kidney and heart and iron overload. #ven thou!h a G6PD Deficient person may not have a crises when consumin! these foods they should be avoided. •
Fava Beans and other legumes $his list contains every le!ume we could find but there may be other names for them that we do not know about. "ow level hemolysis is hemolysis is very hard to detect and can cause other problems so we recommend the avoidance of all le!umes.
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Sulfites and foods containin! them. Sulfites are used in a wide variety of foods so be sure to check labels carefully. carefully.
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Menthol and foods containin! it. $his can be difficult difficult to avoid as tooth paste candy breath mints mouth wash and many other products have menthol added to them. %int from natural mint oils is alri!ht to consume.
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Artificial blue food coloring other artificial food color can also cause hemolysis. &atural food color such as found in foods like turmeric or !rapes is okay.
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Ascorbic acid 'rtificial ascorbic acid commonly commonly put in food and and vitamins can cause hemolysis in lar!e lar!e doses and should be avoided. t is put into so many foods that you can be !ettin! a lot of 'scorbic 'cid 'cid without realizin! realizin! it. See See 'scorbic 'cid 'cid for more information about iron absorbtion and ascorbic acid.
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Vitamin $his is from dru!bank *%enadione (vitamin +,) which is n ot used as a nutritional supplemental form of vitamin + for humans has been reported to cause adverse reactions includin! hemolytic anemia. "ar!e doses have also been reported to cause brain dama!e. -itamin + administered to newborns with G6PD Deficiency has been known to cause adverse outcomes includin! hemolytic anemia neonatal brain or liver dama!e or neonatal death in some cases.*
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Tonic !ater (contains uinine a contraindicated dru! which causes hemolysis in G6PDD people).
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Bitter Gourd and Garden "gg /itter Gourd is also known as /itter %ellon. $hese are common foods in some parts of 'frica and 'sia.
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Some #hinese herbs particularly 0hizoma 1optidis (huan! lien) 1alculus /ovis (neu huan!) 2los 1himonanthi Praecocis (leh mei hua) 2los "onicerae (kam n!an fa) and %ar!arita or anythin! containin! them. #ontraindicated Substances for G6PD Deficient Patients See !6pddeficiency.or! for more information.
'void moth balls artificial blue food color le!umes (fava beans beans peas peanuts soy lentils etc.) sulfites tonic water. Please read all labels on processed foods before feedin! to patient. Substances labeled "ow 0isk %'3 be !iven safely at normal therapeutic doses 4&"#SS the patient is severely G6PD Deficient or has chronic non5spherocytic hemolytic anemia. $here is no !uarantee that low risk dru!s will not cause hemolysis. Please see !6pddeficiency.or! for more information. ' diet rich in antioidant foods is recommended to help minimize the effects of oidative stress and the resultant hemolysis. -itamins b6 b78 folic acid and &'1 are also important. 'cetaminophen 5 "ow 0isk 'cetanilid 5 9i!h 0isk 'cetylphenylhydrazine 5 9i!h 0isk 'minophenazone 5 "ow 0isk 'ntazoline 5 "ow 0isk 'ntipyrine 5 "ow 0isk 'scorbic 'cid 5 "ow 0isk 'spirin 5 9i!h 0isk 'stemizole 5 0isk /eta5&aphthol 5 9i!h 0isk 1hloramphenicol 5 9i!h 0isk 1hlorouine 5 9i!h 0isk 1iprofloacin 5 9i!h 0isk 1olchicine 5 "ow 0isk Dapsone 5 9i!h 0isk Dimercaprol 5 9i!h 0isk Diphenhydramine 5 "ow 0isk Dopamine 5 "ow 0isk Doorubicin 5 9i!h 0isk #thanol 5 9i!h 0isk 2urazolidone 5 9i!h 0isk 2urosemide 5 9i!h 0isk Gadopentetate dime!lumine 5 9i!h 0isk Glucosulfone 5 9i!h 0isk Glyburide 5 9i!h 0isk 9enna 5 9i!h 0isk buprofen 5 "ow 0isk sobutyl &itrite 5 9i!h 0isk soniazid 5 "ow 0isk "amotri!ine 5 9i!h 0isk "evofloacin 5 9i!h 0isk "isinopril 5 hi!h 0isk %a!nevist 5 0isk %eflouine 5 9i!h 0isk %enadiol Sodium Sulfate (-itamin k< sodium sulfate) 5 9i!h 0isk %enadione 5 9i!h 0isk %enadione sodium /isulfite (-itamin +, sodium bisulfite) 5 9i!h 0isk %enthol 5 9i!h 0isk %esalazine 5 9i!h 0isk %etformin 5 9i!h 0isk %ethylene /lue 5 9i!h 0isk %irtazapine 5 "ow 0isk %oifloacin 5 9i!h 0isk &alidiic 'cid 5 9i!h 0isk &aphthalene 5 9i!h 0isk &imesulide 5 9i!h 0isk &iridazole 5 9i!h 0isk
&itrofurantoin 5 9i!h 0isk &itrofurazone 5 9i!h 0isk &orfloacin 5 "ow 0isk :idase 4rate 5 9i!h 0isk Pamauine 5 9i!h 0isk Para5'minobenzoic 'cid 5 "ow 0isk Pefloacin 5 9i!h 0isk Pentauine 5 9i!h 0isk Phenacetin 5 9i!h 0isk Phenazopyridine 5 9i!h 0isk Phenylbutazone 5 "ow 0isk Phenylhydrazine 5 9i!h 0isk Phenytoin 5 "ow 0isk Primauine 5 9i!h 0isk Probenecid 5 9i!h 0isk Procainamide 5 "ow 0isk Pro!uanil 5 "ow 0isk Pyrimethamine 5 "ow 0isk ;uinacrine 5 9i!h 0isk ;uinidine 5 "ow 0isk ;uinine 5 "ow 0isk Stibophen 5 9i!h 0isk Streptomycin 5 "ow 0isk Sulfacetamide 5 9i!h 0isk Sulfacytine 5 "ow 0isk Sulfadiazine 5 "ow 0isk Sulfadimidine 5 9i!h 0isk Sulfafurazole 5 9i!h 0isk Sulfa!uanidine 5 "ow 0isk Sulfamerazine 5 "ow 0isk Sulfamethoazole 5 9i!h 0isk Sulfamethoypyridazine 5 "ow 0isk Sulfanilamide 5 9i!h 0isk Sulfapyridine 5 9i!h 0isk Sulfasalazine 5 9i!h 0isk Sulfathiazole 5 9i!h 0isk Sulfonylurea 5 "ow 0isk Sulfoone 5 9i!h 0isk $amsulosin 5 9i!h 0isk $iaprofenic 'cid 5 "ow 0isk $oluidine /lue 5 9i!h 0isk $riheyphenidyl 5 "ow 0isk $rimethoprim 5 "ow 0isk $rinitrotoluene 5 9i!h 0isk $ripelennamine 5 "ow 0isk -itamin +7 5 "ow 0isk
G6PD Deficient Diet Suggestions G6PD Deficiency can cause or contribute to the metabolic issues discussed belo! !hich can be lessened through $ro$er diet. /y bein! aware of these issues you can make better
informed decisions about the food you eat. $he main ob=ect of the G6PD Deficiency diet should be to obtain necessary nutrients !ith as little use of $recious G6PD (necessary for life) as possible. $his is especially true for people with 1lass 7 (the most severe) G6PD Deficiency. >e can help our bodies conserve G6PD by eating antio%idants& $roviding $lenty of suitable fats and eating fe!er refined carbohydrates . >e need to eat foods that help our bodies repair oidative dama!e and replace dama!ed red blood cells. >e should also be dili!ent in avoiding contraindicated drugs& foods and other substances. :nce you understand the ideas presented below providin! a proper diet for the G6PD Deficient is fairly strai!ht forward and can be uite satisfyin! delicious and full of variety. 'nability to fight o%idative stress( Since G6PD is necessary for the body to produce reduced !lutathione (the body?s bi! !un antioidant) red blood cells are susce$tible to damage by o%idative substances. $his can be minimized by eating a diet rich in antio%idants . "ist of 'ntioidants. Difficulty digesting fats( $his is especially a problem with cholesterol in some who are G6PD Deficient. #holesterol is necessary for $ro$er nerve health and without it we can develop nerve problems such as Peripheral &europathy or %ultiple Sclerosis. f you have a problem with low cholesterol you need a diet rich in fat. :ur fats should come from traditional sources (olive oil& animal fat& $alm oil and coconut oil) and other ve!etable fats should be avoided. "ist of foods and their cholesterol content sorted from hi!hest to lowest.. "ist of foods and their cholesterol content sorted alphabetically. Problems !ith artificial vitamins causing or contributing to hemolysis( )ur vitamins should come from our food as much as $ossible( f supplementation is needed use natural vitamins. >e need to eat a variety of ve!etables especially !reen leafy ve!etables such as lettuce cabba!e spinach kale etc. %ost should be supplemented with the followin! vitamins (dose is for an adult) /7 (thiamine) @A m! daily /, (niacin as niacinamide) @A m! daily /6 (pyridoine) @A m! daily 2olic acid
$ests can be done to check for adeuate levels of vitamins. #at liver and !ood homemade bone stocks (use to make soups and sauces) re!ularly. 'ron( :ne of the problems with hemolytic anemia is that iron is released into the blood stream and can build up to a harmful level. ron can even be fatal if the levels are hi!h enou!h. 1onseuently iron supplements should &#-#0 be !iven to a G6PDD person without testin! for iron levels and should only be done with a Doctor?s supervision. G6PDD people should have their iron levels tested re!ularly as iron overload can cause heart and liver problems. #arbohydrates( Since di!estion of carbohydrates (especially refined su!ar white flour hi!h fructose corn syrup etc) reuires G6PD products containin! these in!redients should be limited. Sulfites( :ur bodies cannot convert sulfites to a usable form therefore sulfites should be avoided.Sulfur is also necessary for a healthy body and should be obtained from sulfates which can be found in garlic and onions& as !ell as "$som Salt baths . *ist of *egumes for Those +ith Glucose,6,Phos$hate Dehydrogenase -G6PD. Deficiency and Favism
2ava beans are contraindicated for people with G6PD Deficiency however many people also react to varyin! de!rees to many or all le!umes. %any times the reaction is not a full blown hemolysis where hospitalization is reuired but does cause hemolysis of a lesser de!ree. :ver time these smaller hemolysis events can lead to other more serious complications or related disorders. See >hy >e 0ecommend 'voidin! "e!umes for more information. $his list is provided as an aid to those wishin! to abstain from all le!umes. f you would like more information about le!umes and !6pd deficiency Plants :f "ife Plants :f Death has in interestin! discussion. Beans
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aduki bean adzuki bean anasazi beans appaloosa bean asuki bean azufrado bean azuki bean baby lima bean bayo bean ben!al bean black azuki bean black bean black turtle bean bolita bean bonavist bean borlotti bean /oston bean /oston navy bean broad bean brown speckled cow bean buffalo bean butter bean butterscotch calypso bean calypso bean canaria bean canario bean cannellini bean chestnut lima bean chili bean 1hristmas lima bean cabeca5de5frade chiporro coco bean52rench white bean coco blanc bean52rench white bean crab eye bean 1ouha!e 1owa!e 1owha!e 1owitch cranberry bean dermason bean Dolichos pruriens edamame #!yptian bean #!yptian white broad bean #n!lish bean #uropean soldier bean eye of the !oat bean faba fa!iolo romano fava bean fava5coceira fayot fazolia bean fei=ao bean feve field pea fla!eolet fool foul fri=o bola ro=a fri=ole ne!ro 2u=i mame ful !reat &orthern bean
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%ada!ascar bean maicoba bean maine 3ellow eye mayocoba bean marrow bean mauritius bean %eican black bean %eican red bean molasses face bean mort!a!e lifter bean mort!a!e runner bean moth dal mucuna bean mucuna pruriens mucuna prurita mun! bean mun! pea mun!o bean navy bean nescaf � orca bean pea bean pearl haricot Peruano bean Peruvian bean picapica pink bean pinto bean p� de mico prince bean purple appaloosa bean ra=ma rattlesnake bean red ball bean red bean red eye bean red chori red kidney bean red :riental bean rice bean rosecoco bean roman bean runner bean salu!!ia salu!ia bean scarlet runner bean Setae Siliuae 9irsutae shell bean small red bean small white bean soy bean soya bean soybean Spanish black bean Spanish $olosana bean speckled brown cow bean Steuben yellow bean Steuben yellow eye bean Stizolobium pruriens Sweet bean Swedish brown bean tapary bean tepary bean $iensin red bean $olosana bean
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!reen !ram haba habas haricot blanc bean horse bean hyacinth bean itchy bean ndian bean Backson wonder bean Bacob?s cattle bean krame kidney bean lablab bean lima bean lin!ot bean
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lupini bean
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ton!ues of fire bean tremmocos trout bean turtle bean turtle soup bean vallarta bean val velvet bean wa bean whit bean white kidney bean white pea bean >indsor bean 3ankee bean yellow ndian woman bean
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yin yan! bean
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Sna$ Beans
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aspara!us bean aspara!us pea bodi boonchi chepil 1hinese lon! bean dau !ok dow !ok dra!on ton!ue bean 2rench bean 2rench !reen bean four5an!led bean !oa bean !reen bean haricot verts talian flat bean
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lon! bean
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manila bean princess pea romano bean runner bean sator snap bean strin! bean $hailand lon! bean wa bean win!ed bean win!ed pea
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yard5lon! bean
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"dible Pods
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1hinese pea pod 1hinese pea 1hinese snow pea
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edible5podded pea
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man!e5tout pea snow pea su!ar pea
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su!ar snap
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Bean Products • • •
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black beans in salted sauce black salted fermented bean 1hinese black bean
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fermented black bean fri=oles refritos refried beans salted black bean
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salty black bean
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dow see
*entils • • • • • • • • • •
arhar arhar dal belu!a black lentil belu!a lentil /en!al !ram black belu!a lentil black chickpeas black !ram black lentil brown lentil
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lablab beans lentilles du Puy lentilles vertes du Puy masar masar dal masoor masoor dal matki moath moon! dal
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channa dal chana dal chilke urad chowli dal continental lentil dal daal dhaal dhal dhall #!yptian lentil 2rench !reen lentils German lentil !ram dal !reen lentil horse !ram ndian brown lentil kala channa
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kali dal
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mussoor mussoor dal petite belu!a lentil Puy lentil red lentil toor toor dal tuvar tuvar dal tur tur dal urad dal val dal white lentil
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yellow lentil
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Peas
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/en!al !ram black5eyed pea black5eye bean black5eye pea black5eyed suzy ceci bean cici bean 1hina bean chawli chickpea chick5pea chole con!o pea con!o bean cowpea crowder pea dried peas #!yptian pea field peas fresh peas !andules !arbanzo bean !arbanzo pea !arbonzo bean !oon!oo pea !reen pea !reen matar dal !reen split pea !un!a pea
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!un!o pea
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kabuli channa kabli chana kabli channa lobhia locust bean lombia no5eyed peas pi!eon pea pois chiches poor man?s pea Southern pea white chickpea yellow pea yellow matar dal
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yellow5eyed pea
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Soy Products • • • • • • • • • •
abura5a!e abura!e aka miso akamiso atsu5a!e atsua!e bamboo yuba barley miso awase miso bean cheese
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plant protein preserved bean curd pressed tofu protein crumbles red miso re!ular tofu roasted soybeans sendai miso shinshu miso shiro miso
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bean curd bean curd sheets bean curd skins bean curd stick bean paste bean sauce bean stick brown rice miso 1hinese yuba dark miso deep fat fried tofu deep5fried tofu doufu dow fu kon dried bean curd stick dried bean stick etra5firm tofu fermented bean cake fermented bean curd fermented soy cheese firm tofu foo yu fried bean curd fu =ook pei fu yi fu yu !enmai miso hat5cho miso hatcho miso inaka miso inaria!e kinu5!oshi kirazu kyoto shiro miso mame miso mamemiso medium tofu mellow white miso miso mu!i miso nama5a!e nama nori san nato natto nattou ni!ari tofu
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okara
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shiromiso silken tofu soft tofu soy cheese soy mayonnaise soy milk soy milk skins soy sour cream soy nuts soy yo!urt soya cheese soya mayonnaise soybean curd soybean paper soybean paste soynuts soy nut butter soynut butter sui5doufu sweet miso sweet white miso tempe tempeh tetured soy protein teturized soy protein tetured ve!etable protein teturized ve!etable protein tofu tofu mayonnaise tofu sour cream $SP $-P uba unohana usu5a!e usua!e ve!etable protein wet bean curd white miso yellow miso
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yuba
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Vegetable Gum Thic/eners These are either made from legumes& or can be made from legumes • • • • • • •
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albumin 5 from peas 'cacia !um carob bean !um 2lavorin! or natural flavorin! !um arabic !uar !um lecithin locust bean !um
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%onosodium Glutamate (from soy) $ara seed !um tra!acanth -e!etable broth (soy or even fava beans) ve!etable emulsifier ve!etable !lycerin -e!etable !elatin
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ve!etable stabilizer
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)ther *egumes •
'lfalfa sprouts
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0ooibos
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'stra!alus (herbal medicine) 1arob (chocolate substitute) 2enu!reek Bicama "icorice
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Peanuts
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0ed $ea 'frican 0ed $ea Senna or 1assia Sin!kamas $amarind -etch 2amily (&ot normally used for food)
)ther Foods *i/ely to #ontain 0idden Soy or *egume Additives • • • • • • • •
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'rtificial butter flavor /aked !oods 1andies 1anned meats or tuna 1anned soups 1hips 1hinese food Gravy %ies
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"ow fat cheeses or cheese substitutes %ar!arine Sausa!es hot do!s processed meats Sauces (>orcestshire. Sweet and Sour etc) Salad Dressin!s Stock or bouillon $ofutti
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Powdered foods
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nfant formula
Why We Recommend Avoiding Legumes There is a lot of controversy about legumes causing hemolysis in G6PD Deficient patients. In order to address this issue, I will first address the issue of varying degrees of hemolysis.
Low Level Hemolysis Most doctors and other medical professionals see hemolysis in G6PDD patients as an all or nothing problem. If you dont hemoly!e badly enough to send you to the hospital, youre fine. I strongly disagree with this for the following reasons" •
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If that were true, all hemolytic events would end in death, and this is not correct. The vast ma#ority of hemolytic events are mild enough for the body to compensate for without intervention. $ollowing this reasoning, it is only logical that hemolysis can happen from very mild to very severe, depending on circumstances such as health, stress, trigger, age, etc. Many people go for years e%periencing hemolysis without &nowing it. They can have other health issues that eventually lead to the discovery that they have G6PDD, or they eventually have a hemolytic crises. I have received countless emails from people in this category. Their health issues run from liver, heart, blindness, renal, spleen and chronic yellow color to s&in, to death in some cases. These problems can occur from early in life to later in life. Many families discover G6PDD runs in their family only after the needless death or serious illness, caused by G6PDD complications, of a family member.
What Causes Hemolysis 'ow that we &now that hemolysis varies in intensity, lets discuss the cause of hemolysis. (hen a red blood cell comes into contact with an o%idative substance, an )*+ with sufficient G6PD to reduce glutathione will neutrali!e the o%idative substance rendering it harmless. Those with G6PDD cannot reduce enough glutathione to protect )*+s from damage so, the o%idative substance destroys the )*+. It is my opinion that this happens to everyone with G6PDD, regardless of which variant they have. (hat is more important than variant is the degree of G6PDD the person has. ne person may have more G6PD than another so that person is able to produce more reduced glutathione to protect )*+s than a person with less G6PD. $or the purpose of this discussion both less G6PD and less effective G6PD are considered the same.
Legumes and Hemolysis 'ow I will discuss legumes. In every contraindicated list I have ever seen, fava beans, or broad beans, are considered contraindicated for all variants of G6PDD, yet some insist that all variants of G6PDD do not e%hibit favism. The definition of favism is a condition that causes hemolysis from e%posure to fava beans. -s of now I have never seen a research paper or other proof as to the e%act chemical, or chemicals, in fava beans that cause hemolysis. ver the past few years, some people using hemoglobin meters have shown that many other legumes also cause hemolysis to varying degrees. -gain, it is my opinion that all people react to these substances, but to varying degrees depending on severity of G6PDD, health, etc., as described above. *ecause low level hemolysis or mild hemolysis/ is very hard to detect, it is logical that many people believe that they are not reacting to legumes or other substances that cause low level hemolysis. *ut, low level hemolysis can be very dangerous over time. ur bodies must generate more )*+s to
compensate for the ones that are destroyed and the destroyed )*+s must be cleaned up. This process ta&es resources needed for healthy bodies, conse0uently we are more susceptible to other diseases and they can be more severe than when we are not undergoing low level hemolysis. Medical research is far behind when it comes to legumes and G6PDD. *ecause I have had so much success in stopping hemolysis by avoiding all legumes and products containing them, I recommend that they be avoided. 1opefully, someday maybe medical research will provide us with more information concerning the e%act chemicals they contain that causes hemolysis.
This from a doctor. $ellow travelers" 1ow much rat poison do you need in your system to poison your body2 1ow often can you e%pose yourself to it to become immune2 If 344 people are e%posed to poison, do all die or bleed overtly2 -nd, those who show no clinical signs of poison are they assumed not harmed in any way2 It is funny, once you consume poison regardless of clinical presentation, you get treated because we &now in the hospital that damage is done regardless. Poisons are dangerous to humans because we lac& the specific en!ymes to brea& them down into non5threatening wastes. i&ewise, o%idative stressors are our &ryptonite. 7uperman had enough sense to not only stay away from it but bury it out of his e%istence. 1e didnt ta&e any chances. 'on of us is super person so we must pay heed. 'ot &nowing the facts is one thing, but to be e%posed to facts and not consider2 That is another thing. There seems to be such arrogance in ignorance that it is baffling. 8uite a big difference between allergy and en!ymopathy. -llergy involves the immune system and the body can be re5trained in a method called de5sensiti!ation. n the other hand, en!yme deficiency or dysfunction is what it is, a lac& in functionality. (hen a deficient body is e%posed, there is untoward ramification. 7een or unseen the damage happened. 9ven, the so called partial deficient female I call insufficiency/ definitely suffers damage as well when e%posed. 1owever, the response in her case could be li&ened to a double engine plane in crisis. (hen one engine is done, unli&e its single engine counterpart, it continues to fly and land with the other. -s&, any seasoned pilot, theyll attest that it is not the most desirable situation and that plane got a problem. -s I read along it seems that no matter what li&es of Marion, Dale, :ohn and many others advocate, there will be people who prefer to bury their heads in the sand. There is a cliche that g6pdd is not a serious problem. +liches are myths and li&e all myths based on pre#udicial ignorance and non5facts. The damning danger is that the group most guilty of perpetuating this myth is no other than those sworn &eepers of our health, our physicians. This is not only the irony but a disgrace for their lac& of investigative curiosity in this genetic dysfunction. Imagine, this is not only one of the ; most common newborn genetic disorder but, causes more &ernicterusbrain damage due to high bilirubin/. male siblings two same day/ after being e%posed to a stressor. My vigilante rescued me from my moms fate when my oldest son too& ill after e%posure to mothballs. Ironically, my little study has shown that most babies with g6pdd do not naturally li&e beans but are taught or forced into ac0uiring the taste. 'ature always has a way of protecting us if we pay heed. *e well. Dr. gundu
