Ti ook otai th opiio ad ida o th autho. It i itdd to povid hlpul gal iomatio o th ujt that it add. It i ot i ay way a utitut o th advi o th ad’ ow phyiia() o oth mdial poioal ad o th ad’ ow idividual oditio, ymptom, o o. I th ad d poal mdial, halth, ditay, xi, o oth aita o advi, th ad hould oult a omptt phyiia ad/o oth qualid halth a poioal. T autho ad pulih pially dilaim all poiility o ay ijuy, damag, o lo that th ad may iu a a dit o idit oqu o ollowig ay ditio o uggtio giv i th ook o patiipatig i ay pogam did i th ook.
scrIbner A Diviio o simo & shut, I. 1230 Avu o th Amia nw Yok, nY 10020 copyight © 2013 y sua blum, M.D. All ight vd, iludi iludigg th ight to podu thi ook o potio tho i ay om whatov. Fo iomatio add si suidiay right Dpatmt, 1230 Avu o th Amia, nw Yok, nY 10020. Fit si hadov ditio Apil 2013 s c r I b n e r ad dig a gitd tadmak o
T Gal Goup, I., ud ud li y simo & shut, I., th pulih o thi wok. Fo iomatio aout pial diout o ulk puha, pla otat simo & shut spial sal at 1-866-506-1949 o ui@imoadhut.om.
T simo & shut spak buau a ig autho to you liv vt. Fo mo iomatio o to ook a vt otat th simo & shut spak buau at 1-866-248-3049 o viit ou wit at www.imopak.om. Mauatud i th Uitd stat o Amia 1
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Liay o cog catalogig cotol num: num: 2012031929 20120319 29 Isbn 978-1-4516-9497-0 Isbn 978-1-4516-9500-7 (ook)
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FOreWOrD y Mak Hyma, M.D. InrODUcIOn
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chapt 1: Autoimmu Dia Dia bai
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USING FOOD AS MEDICINE chapt 2: Uig Food a Mdii chapt 3: Uig Food a Mdii Wokook chapt 4: Ui chapt Uigg Food a Mdii rip
37 60 91
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UNDERSTANDING UNDERST ANDING THE STRESS CONNECTION chapt 5: Ud chapt Udtadig tadig th st cotio chapt 6: Udtadig th st co cotio tio Wokook chapt 7: Ud chapt Udtadig tadig th st cotio rip
105 129 158
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HEALING YOUR GUT chapt 8: Halig You Gut chapt 9: Halig You Gut Wokook chapt 10: Halig You Gut rip
175 191 215
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SUPPORTING YOUR LIVER chapt 11: suppotig You Liv chapt 12: suppotig You Liv Wokook chapt 13: suppotig You Liv rip
229 257 280
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ADDITIONAL CONSIDERA CONSIDERATIONS TIONS chapt 14: Itio ad spi Autoimmu coditio coditio
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cOncLUsIOn Akowldgmt Appdix I: Hlpul book, cD, At-Hom Pogam, ad Dtiatio Pogam Appdix II: supplmt ad H Guid not Additioal r r rip Idx Idx
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FOREwORD
rlmaHddepdm PaadSfg What dia at mo wom tha hat dia ad at a omid? What polm polm at 24 millio Amia ut iv oly 5.4 pt o th natioal Ititut o Halth’ (nIH) udgt o thi polm to tudy it pottial au? T aw i autoimmu dia. Ad it i motly o th ada au it om i o may avo ad typ. rhumatoid athiti, lupu, multipl loi, iammatoy owl dia o oliti, diat, hypothyoidim, ad poiai a all autoimmu dia. W thik o all th polm paatly, wh thy a ally jut o dia with may dit dit olo, dpdig o you ag, x, ad g. Autoimmu dia at ou odi at almot vy lvl. som at ou vou ytm—lik autim ad may v dpio—ad oth at ou joit ad mul, ou ki, ou doi glad, ou hat, ad mo. Autoimmu dia ou wh th ody’ immu ytm attak it ow tiu ath tha a oig molul, uh a a atia. T a mo tha o hudd dit autoimmu dia, ad a ayo livig with a autoimmu oditio will tll you, it ha a hug impat o quality o li. T mgig i—ad it patial appliatio though utioal mdii—poit to jut a w ai au o autoimmu autoimmu dia, mot o whih a igod y ovtioal doto, who ty to hut o th immu ytm with powul mdiatio that hav dagou id t. but th tuth i that th oot au o almot all autoimmu dia a th am: mio, viomtal viomtal toxi, allg, t, ad poo dit. by movig th oot au ad uppotig th ody’ opti-
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mal utioig though dit ad lityl, you a atually ov om th oditio oditio. . D. sua blum’ goudakig ook, Te Immune System Recovery Plan, i a powul guid to l-halig. D. blum how how you a add th oot au o dia ad ala you immu ytm. sh lay out a la oad map to ovy o th millio o popl dlly uig om autoimmu dia ad povid olutio o hag i dit, upplmt, ad viomt that a hlp popl dal with ad v v autoimmu oditio. D. blum’ di to d th aw wa uld y h ow tuggl tugg l with autoimmu autoimmu dia, a oditio h ha uully tatd ad ovd om y uig th pogam did i thi ook. Ou ovtioal appoah to autoimmu dia i to hut dow th immu po with powul mdiatio iludig nsAID lik Advil o Alv, toid lik pdio, atia dug lik mthotxat, ad th w dug lik el, Humia, ad rmiad that lok th t o a powul iammatoy iammatoy molul alld nF alpha. but tho w dug low dow you immu ytm o powully that thy ia you ik o a o li-thatig li-thatig itio. Ad thy hav qut ad iou id t ad o giv oly patial li. T dug may liavig o om i th hot tm, ut i th log tm thy do nothing nothing to to add th au o dia. D.. blum ad I u th piipl o utioal mdii, whih tak D u to th oot o th polm. W hav tatd hudd o patit with autoimmu dia uully y addig th udlyig au, iludig toxi, toxi, itio, allg, poo dit, ad t. I hav v td om th mthod myl. choi choi atigu ydom ha autoimmu atu ad my lood tt laly howd my ody wa attakig itl. Gttig id o my muy poioig vd my hoi atigu ad autoimmu polm. Ad thi ha tu o o may patit. Fo ah o, doto lik D. D. blum ad I d all th au—toxi, allg, itio, poo dit, ad t—ad dal with all o thm whil addig ak th thig th ody d to utio optimally—whol, la ood, utit, xi, t maagmt, la wat ad oxyg, ommuity, otio, ad maig. H a om o th toi o ovy that a poil o you. •
O t-ya-old gil am to m with mixd otiv tiu dia—h ki, joit, liv, ad lood ll w all avagd y
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iammatio. Doto attmptd ut aild to otol h ymptom with ho do o itavou toid ad immu-uppig hmo dug. no o akd “why.” Why wa h iamd? What aud th ovativity o h immu ytm? Ty imply watd watd to hut it dow. A two moth o a glut- ad daiy- dit, laig out all th yat i h gut ad toig h utitio with whol ood ad upplmt, upplmt , h wa damatially damatia lly tt. A a ya, ya, h had o ymptom ad wa o all h mdiatio ad h autoatiodi tud to omal. Ti typ o dia val i ot i ovtioal pati, ut it i poil ad availal to vyo ow though D. blum’ ook, Te Immune System Recovery Plan. O woma had ipplig poiai ad latd athiti. sh wa otytwo ad ould’t walk up ad dow tai o gt ito a athtu y hl, o ould h poply a o h hild. ni moth a w tatd tatmt, iludig limiatig glut ad oth ood allg, movig h havy mtal, ad alaig h immu ytm, h walkd ak ito my o ot oly thity poud light (ig iamd mak you at) ut ompltly o pai ad poiai. Aoth ma ud o ya with th loody diaha ad pai o ulativ oliti. Ditay hag ad vaiou digtiv d igtiv uppot uppot hlpd ut h v got tt util w lad out th ad atia om hi gut ad got him o glut. g lut. Ad a t patit patit with dilitatig atigu ad multipl loi ad littl whit iammatoy a o h ai xpid aly omplt li o h ymptom a h had th muy dtal llig movd ad wt o a omphiv dtoxiatio pogam. Ad wh h patd h MrI, all o th whit a om th Ms w go!
Futioal mdii pati aoud th outy a hlpig outl patit to ov om autoimmu dia. D. sua blum i th viioay hid o uh pati, ad at blum ct c t o Halth, h ha impovd th liv o thouad o popl. T appoah o dig ad movig tigg o dia, uh a hidd mio, toxi, o allg, ad uppotig th ody’ ody’ utio with utit, h, ad “po” dug uh a poioti i a movmt that i ow ig patid y patitio at th uttig dg o mdii. It i a appoah alld utioal mdii ad ha hlpd t o thouad o patit woldwid. T valual widom o utioal mdii i did laly ad thooughly i Te Immune System Recovery Plan. by ollowig th
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taightowad, volutioay taightowad, volutioay pogam did i thi ook, you, too, a tat th au o autoimmu dia, v you oditio, ad giv youl th gi o halth ad wll-ig. Futioal mdii giv u th kowldg ad th mthod. now you jut d to appl applyy thm. Mak Hyma, M.D. Wt stokidg, MA sptm 2012
INTRODUCTION
GtgSad AnwPashp T’ a gowig pidmi i ou outy. It do’t ga wpap hadli o mak th vig w, ut it’ th ad it i dilitatig ad pottially dadly. It i th pidmi o autoimmu dia. Autoimmune diseases diseas es are among the most prevalent orms o chronic illness in this country, now aecting an estimated 23.5 million Americans. Mo popl u om thi goup o hoi ill tha om a o hat dia, yt mot popl do’t v kow what th oditio a. Ti lak o awa i killig u—litally. It’ auig v pai, diaility, ad v dath. Wo yt, may popl with autoimmu oditio u with thi ymptom au ovtioal doto ith a a’t ’t gu out what’ wh at’ wog o a a’t ’t gt to th t h oot o th polm. pol m. T aw i utioal mdii, a mdial pialty that ha om o ag i th pat t ya ad ou o vig th hoi oditio. Hop i h. You’ holdig it i you had.
WHY A Book on AuToiMMune DiSeASe? T a mo tha o hudd dit autoimmu oditio, ad thy a all iou hoi dia aud y a udlyig polm i th immu ytm. Ti ook i a all to alam: these diseases are reversible and curable i caught early, beore they progress and cause severe pain, disability, and even death. T t ymptom might vagu lig o atigu, mul o joit pai, o a aggig lig that omthig i’t ight. With impl lood tt, you a diagod at thi aly tag, ad th, y ollowig th tp i thi ook, you immu ytm a ought ak ito ala o ivil damag i do to you ai, joit, thyoid, lood vl, ad oth vital oga. I you hav uig om a autoimmu oditio o om tim ad you hav alady xpid om tiu damag, thi ook will hlp
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you l tt i gal, how how you a atually v th t o th dia, ad hlp pvt you pi tiu damag om gttig wo. A a doto, I hav log awa o autoimmu oditio uh a Gav’ dia, humatoid athiti (rA), coh’ dia, ulativ oliti, lupu, multipl loi (Ms), Hahimoto’ thyoiditi, poiai, alopia aata, vitiligo, sjög’ ydom, ad lodma, amog oth. but wh I wa diagod with o o th ill, it hagd oth my poal ad poioal liv ov.
MY STorY bo I go ay uth, lt m itodu myl th way I would i you w o o th patit i my o. I’m D. sua blum, a oadtid phyiia i pvtiv mdii ad a aitat liial poo i th Dpatmt o Pvtiv Mdii at th Mout siai shool o Mdii i nw Yok city. I am alo th oud o blum ct o Halth i ry book, nw Yok. I mdial hool, I tatd dow th oad o taditioal mdii. T duig my ital mdii idy, I quikly alizd that I did’t wat to ou jut o ill o dia; I watd to ou o preventing tho venting tho oditio i th t pla. but ak th pvti pvtiv v mdii oitd motly o ig tt ad puli halth pogam. It wa’t pat o a doto’ vyday pati. Phyiia jut w’t giv th tool i mdial hool to hlp thi patit hag thi havio, uh a how to gt popl to at halthully o du t. I kw I had to tak a dit out a a doto, o I ought taiig om ititutio that w oidd “outid th ox” at th tim. I patiula, I wa ittd i th latiohip tw t, utitio, ad ill. I movd o th at path o taditioal mdii towad a mo holiti appoah ad ito a mgig ld alld utioal mdii. Fit I ompltd a pogam at th ct c t o Mid-body Mdii i Wahigto, D.c., wh I lad tool o t maagmt ad mthod to v th t o t o th ody. T, at th Ititut o Futioal Mdii, Mdii, I lad how ood at th th po, ith pomotig halth o auig dia. Wh I tatd applyig what I had lad aout t maagmt ad utitio i my ow pati, I aw thad how th mthod hlpd popl to pvt ad atually reverse atually reverse hoi dia. T two ida—pvtio ad val o hoi dia—am my paio ad a at th o o
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my mdial pati today. but th i o aa wh th thiqu a tuly li-hagi li-hagig: g: autoimmu dia. I kow thi ot oly au I hav th t o my patit who u om autoimmu autoimmu dia ut alo au I aw th impat o myl wh I wa diagod with a autoimmu autoimmu dia. I diovd it mo tha a dad ago wh a id akd m why my had w o yllow. I atually had’t otid thi o, ut h wa ight, o I immdiatly had lood wok do to d out why. “You hav hypothyoidim,” my doto told m a th la ult am ak. Me? ak. Me? A A thyoid polm? I ould’t ould’t liv li v it. Hypothyoidim i a oditio i whih th thyoid glad do’t podu ough thyoid homo. homo. Ti homo i ay to hlp th ody ody ovt taaot, a utit oud i yllow ad oag uit ad vgtal, ito vitami A. bau thi ovio wa’t happig, all th taaot om my halthul dit wa wa ’t ig poply pod. Itad, th utit w uildig up i my ody, ad o ymptom o thi wa my yllow had. I topt, I alizd that thi wa’t th oly o. Aoth wa th at that I had to wok ally had to kp om gaiig wight, wight, ad a thid wa that I wa o vy tid. but at th tim I did’t aliz th w ig o a polm. Ty had pat o my li o o log that I oidd thm “omal.” Futh ttig vald v mo ad w. I had a autoimmu dia alld Hahimoto’ thyoiditi. I oth wod, my immu ytm, whih i omally omal ly th ody’ ody’ d i th daily attl agait itio ad ivad, wa o log pottig o ddig m. I at, my immu ll had tud agait my thyoid, attakig it ad damagig it to th poit wh it wa tugglig uuully to mak ough thyoid homo. I wa hokd. I had om a vgtaia ya ali, I xid gulaly, I patid yoga ad mditatio, ad I had ulti vatd a tog, piitual aith. I had ad my dmo i pyhotha pyhothapy py ad wa ally ott with my li. bau I wa doig what I thought w all th ight thig alady, ig diagod with a autoimmu dia wa hokig ad ightig. I wa alo a mdial doto, o I jut ould’t ould’t liv that omthig omthig wa goig o iid my ody ody that I did’t did ’t kow aout. a out. but my pimay a phyiia jut huggd o thi upttig w. “It’ o ig dal,” h aid. “You’ll jut tak thyoid homo plamt mdiatio ad .” .” no ig dal? d al? May ot to him, ut my ituitio told m omthig wa’t wa’t ight. My ody wa out o ala, ad I watd aw. Why did I I hav hav thi dia? Ad why now why now?? o udtad my
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ill, I didd to u it a a oppotuity to xplo all that taditioal ad otaditioal mdii had to o m. I didn’t want to take a pill that would only mask the problem and just manage my symptoms. I wanted to know why know why those those symptoms were there in the rst place.
HoW FuncTionAL MeDicine HeLPeD Me T jouy that I ga a my diagoi o Hahimoto’ dia ld m dp ito th uiqu ad, at th tim, lativly w ld o utioal mdii, a appoah that oid th whol po, ot jut hi o h ymptom. A ivig my diagoi, I ga uig th piipl I lad at th Ititut o Futioal Mdii to tat myl. A a ult, I diov diovd d that au au o a gti pdipoitio, pdipoitio, I’m at high ik o autoimmu dia. I alo lad that my ody ha toul movig muy om my ytm. (Muy i a toxi that’ damagig to tiu i ou odi ad a pottial au o autoimmu dia.) I wt o a glut- dit ad dud my muy xpou y o log atig h high i thi havy mtal (uh a tua, wodh, l, ad tipd a), ad ov th ou o two ya I had my may dtal amalgam llig movd. I ga to dik poti hak that du iammatio ad ha th utio o th liv. (si th liv i th ody’ mai dtoxiyig oga, thi hlp it la muy ad oth toxi om th ody.) wo ya a I lad I had Hahimoto’ ad tatd th jouy j ouy to tat my autoimmu autoimmu dia uig utioal mdii, my atiody lvl w omal ad my dia wa ud. I wa takig a mall amout o thyoid homo (alld lvothyoxi), ad I had gat gy ad my wight wa ay to maitai. I wa thilld thil ld ad xitd to ha what I had lad with my patit. Tat wa th t tp. bau my thyoid wa alady damagd wh I wa t diagod, my xt goal wa halig my thyoid, whih a tak a vaial amout o tim o ah po, dpdig o how log it ha ud attak y th autoimmu dia. si my thyoid glad wa quit damagd om th Hahimoto’, I till dd, ad otiu to d, thyoid homo plamt. Tat aid, I ow tak hal th do that I wa takig t ya ago. oday I l halthi ad mo gti tha I v hav. My halth jouy ha taught m what my ody d, how to ak, ad how to lit. Reversing a chronic disease takes time and eort, but it can be done. My toy i poo o that. I did it o myl ad mad th poioal diio to hlp oth popl livig with autoimmu dia do it, too.
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Ya ago I gav a ltu to a audi ull o halth a poioal at th ct o Mid-body Mdii’ poioal taiig pogam. Duig that ltu, I talkd aout th impota o uig ood a mdii ad how impl thig uh a athig ad laxatio a wok to hlp hal dia. I wa tud to d that th impotat, lihagig opt w w to mot o th two hudd popl i th audi. Ad thy w halth a poioal! I ould’t liv it. How ould th doto tah th thig to thi patit i thy did did’t ’t udtad thm ad w’t patiig thm thmlv? It wa th that I alizd it wa tim to gt th wod out mo oadly tha imply to my patit ad th popl who attdd that ltu ad oth talk I otiu to giv.
HoW FuncTionAL MeDicine cAn HeLP You I 2010, a val ya o plaig (ad may mo pt damig aout uh a oppotuity), I opd blum ct o Halth i ry book, nw Yok, wh th ou i o a omphiv itgatio o utioal mdii, mid-ody mdii, ad pvtiv pvtiv mdii. It ilud a ookig hool to tah patit ad lit how to at o thi halth ad a mid-ody-piit t that tah a aay o laxatio thiqu ot oly to pvt hoi halth oditio ut alo to v thm. Ti, omid with th utioal mdii tatmt I o a a mdial doto, ha atd xtaodiay, li-hagig ult o my patit. I’m ot th oly po who ha told to imply pop a w pill ad mov o. Ad I’m taily ot th oly o who thought that th hould a tt way w ay.. oday oday,, i my utioal u tioal mdii mdi i pati pati at blum ct o Halth, patit o ha with m what thi taditioal mdial doto hav aid a diagoig thm with a autoimmu dia: “Gt ud to th paikill. p aikill. You’ll You’ll d thm o th t o you li,” “W do’t kow why popl gt th dia,” ad “You immu ytm a’t tatd.” T a jut a w xampl, ut th uig thm i gally o o hopl. T ida i that you hould jut apt you diagoi ad th ivital lilog attl with paikill, dug that upp you immu ytm, ad th id t o oth (om o whih a dvatatig ad log-tm). I imply ud to apt that th wa othig I ould do o myl o th oth 23.5 millio Amia utly livig with autoimmu autoimmu dia. but with th t h pval o autoimmu dia o th i, I kow that th a
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may, may popl livig with th oditio who will v mak it to my o. I wot thi ook ook o I ould o thi pla—a tiv ad halthy appoah—to a oad audi. May taditioal doto hav a attitud o “jut giv i to you ill,” whih oly lad you to l uttly powl. It also makes your disease worse because it undermines the body’s natural healing process. Te mind-body connection is real and powerul. T a may tudi that th at how that th mo otol a po l h o h ha ov a ituatio, ituatio , th high th at o halig. Popl do tt, tt, l tt tt,, ad liv tt with vaiou halth oditio i thy hav a that th i omthig thy a do o thmlv. Ad th i! Tat i o o th o piipl o utioal mdii, ad a a xpt i th ld, I’m h to how you how muh otol you a hav ov you ow halth ad wll-ig. so what xatly i a utioal mdii xpt? Fit, lt m tll you what it i ot. Mot taditioal doto tak ot o you ymptom ad gu out what diagoi thy a udl tho ymptom ito. Ti uually ditat you tatmt. To, i you ad a id hav th am ymptom, you o gt th am diagoi ad th th am tatmt. Ti wok o tauma ad o halth iu uh a appdiiti o a ok lg, wh tai thig uh a ugy a mdy a immdiat polm o ymptom. Uotuatly, thi appoah do’t wok wh you’ tyig to tat ad pvt omplx, hoi ill uh a auto autoimmu immu dia. A utioal mdii xpt i lik a mdial dttiv gathig all th lu o you pat (wh you gw up, you amily ituatio, taumati vt, halth hitoy, t.) ad you pt (pottial toxi i you viomt, oial li, t lvl, latiohip, dit, wokout gim, lp hait, ymptom, t.). Amd with thi iomatio, a utioal mdii xpt ti to uov how ad why you ody i ot utioig wll. Ti typ o dttiv wok i what I o all o my patit at blum ct o Halth ad what I ud to u myl o th iou autoimmu oditio that I had. It i xatly what I will o you with th itativ lmt that u thoughout thi ook. sidy bak, M.D., M.D., a wll-kow pvtiv pvtiv mdii pialit p ialit who i o alld th t h ath o utioal mdii, o aid, aid, “I you a ittig o a tak, th aw i ot to tat th pai. T olutio i to d th tak ad mov it.” Te Immune System Recovery Plan will hlp you d th “tak” that a a auig you immu ytm to malutio malu tio ad mov thm o y o. It will tak you though th ou itial tp
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that will mov thig om th ody that a ad o th immu ytm ad th mak u th ody ha xatly what it d to utio poply.
THe iMMune SY SYSTeM STeM recoVerY PLAn I wot Te Immune System Recovery Plan to ha a impotat mag o hop ad halig. Shockingly, there is no other book on the market that tells the average person how the immune system works that’ xatly what I’ll dtail i th pag. p ag. and shows how to x it. Ad that’ I will xplai how I hav hlpd hundreds o popl with autoimmu dia uh a lupu, humatoid athiti, sjög’ ydom, Hahimoto’ dia, ad Gav’ dia, amog oth, v thi ymptom ad du thi atiodi. Atiodi a molul omally mad y you immu ll to attak ad dtoy oig ivad, ut i autoimmu dia, th atiodi a miditd ad attak ad damag you ow tiu. May o th oditio a hoi, dilitatig, ad pottially lithatig. I at, autoimmu dia a o o th top t ladig au o dath i gil ad i wom up to ixty-ou ya o ag. Te Immune System Recovery Plan will hlp you gu out what you’ lig, why why you’ you’ xpiig tai ymptom, ad how you a tat th udlyig au o tho ymptom o you a v you autoimmu dia. How a o ook add whatv autoimmu oditio you hav? bau mot o th oditio hav imila udlyig imala. Tough th itativ apt i Te Immune System Recovery Plan, whih ilud hklit, qutioa qutioai, i, ad lamt, you will al to do with thi ook what my patit do with m i my o. natually natual ly,, you wo’t wo’t doig you ow lood loo d wok o tool tt (i tho t ho qui a halth a poioal), poioal), ut y uig th ou tp i thi ook, you will la how to tat whatv autoimautoimmu oditio you hav. T ou tp to halig ad pvtig autoimmu dia a: •
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stp 1: Uig ood a mdii stp 2: Udtadig th t otio stp 3: Halig you gut stp 4: suppotig you liv
I uggt you do th ou tp i od, whih will hlp you ap th mot t om thi ook. Howv, ah o a alo tad alo, o you
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a tagt o aa at a tim i you p. Te Immune System Recovery Plan i dividd ito ou pat, opodig to th ou tp. eah pat i dividd ito th hapt: •
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T t hapt i ah pat o a clear explanation about how the how the immune system is aected y th topi at had—o xampl, how th immu ytm pod to t o how digtiv halth iu th immu ytm. T od hapt i a workbook with sel-assessments. Uig th ult o th amt, I will hlp you poaliz you ow tatmt pla. Ti hapt will povid all th iomatio you d to implmt implm t th pla, iludig pi itutio o how to itga itgat t th tool ito daily li ad how to mak pmat lityl hag. T thid hapt povid recipes that how you how to u ood a pat o you poalizd tatmt pla. eah ip ilud ood pially tagtd to ah o th ou tp.
A w mov though th ou tp, I will xplai why ah i impotat ad how you how quikly you a gi to l tt. by ollowig ah o th ou tp, ith alo o togth, you will gi to impov you immu ytm immdiatly. T tp a th oudatio that I u i th tatmt pla I at o vy o o my patit ad a th implt ad mot tiv “mdii.” Te Immune System System Recovery Recover y Plan will aw th t h ollowig qutio ad may mo: •
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How th v A’ A’ (ataid, (ataid , atiioti, atiioti , Advil, alohol, alohol , ad aimal ood) oo d) mak you ik Why th mot ommo ood i th tadad Amia dit a ommoly mitak y you ody a ivad, auig you immu ytm to attak you ow ody How xpou to muy ad oth toxi hag th ompoitio o you tiu, auig you immu ytm to attak you ow ody How ommo viu—uh a th epti-ba viu, whih au moo—a povok a ogoig immu atio that au you to l hoially woll, ti, t i, puy, puy, ad tid How ovtioal tatmt, whih hut o th ody’ immu po, hav iou id t iludig iomia, wight gai, mul pai, iad lood pu pu, , ad dpio How th utioal mdii appoah appoah a ud i oju ojutio tio with taditioal tatmt tatmt to o ig t ad vy littl ik
Gtg Sad A nw Pashp •
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How vy viu you’v v had—iludig old o, hik pox, higl, hpatiti, epti-ba, epti-ba, ad mo—la mo—lav v idual patil i you ody that a dvlop ito autoimmu autoimmu dia, ad how to mak u that tho oditio a’t atd i you ody How 70 pt o you immu ytm liv i you gut, ad why alaig th good ad ad atia i you digtiv tat a lad to optimal immu utio How havig th ight amout o atia i you gut du th iid o allgi ad auto autoimmu immu dia
HeLP For uS ALL O thig I’d lik to ot i that although thi ook highlight autoimmu oditio oditio/dia, /dia, Te Immune System Recovery Plan will help anyone wishing to have a healthier, more balanced immune system. T tp outlid h hav hlpd my patit who u om athma ad allgi, a wll a popl who l thy ath vy u o old ug that thy out. Mo popl today tha v o hav autoimmu dia, ad thi pidmi i patially th ult o a viomt viomt that omad u with toxi ood, t, hmial, ad havy mtal. Ad whil w aot liv i a ul, w can pott ou halth y omig mo awa ad hagig what om ito ou hom, ito ou amili, amili , ad ito ou odi. evy igl o o u ha oth a hoi and and aa poiility to a o oulv. What’ mot impotat to kow i that w do hav otol ov what happ to u. Y, being diagnosed with an autoimmune disease is something to take seriously. But it is not a lie sentence and should not be treated as one. You do’t hav to liv i pai o tak dilitatig mdiatio o th t o you li. You a hal you ow immu ytm. I kow thi ot oly au I’v xpid thi with may o my my patit ut alo au I hav livd it. Te Immune System System Recovery Recover y Plan giv you tool o tatig ad v vig you autoimmu oditio a wll a th th tool o pvtig thm i th t pla. My goal o you whil adig thi ook i to that autoimmu diod are tatal ad that pmat olutio do xit yod a litim o takig pill. T is hop ad o muh that you—y, you you—y, you—a —a do to th ailitato o you ow halig. Ad you a ot alo. I am h to guid you o thi jouy, o lt’ gt tatd!
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According to the National Institutes o Health (NIH), “Research discoveries o the last decade have made autoimmune research one o the most promising new areas o discovery.”
BeFore You BeGin I aliz that thi ook otai thial iomatio that a omtim l ovwhlmig ad tatmt pla that a akig you to mak may hag i you dit ad you li. but a you ad though it all, tak a dp ath ad look pat th t. I wat you to th ot. I wat you to kow that th a optio that do’t ivolv mdiatio— optio that a atually hlp you hal you immu ytm. Whil you do d to ou o th t, whih a th dtail o ah o th ou tp i thi ook, o ok, kp you y o th ig pitu o what w a tyig to aomplih: aomplih: halig you ody o th t h dpt lvl. My goal o thi ook ook a thold. Fit, I wat you to aliz that a omou ody o iti litatu uppot thi appoah to halig ad alaig th immu ytm. You may om up agait phyiia who a uamilia with utioal mdii ad uamilia with om o th tudi I ha with you i thi t hi ook. but jut au au you doto do’t kow aout thi iomatio do’t ma it i wog. My goal i thi ook ook i to mpow you to tak hag o you ow halth. I alo wat you to hav th od to ha thi iomatio with oth. My od goal i to o you tatmt lik what w hat you might gt at my o. T a millio o you ad oly o o m. Ti i why I atd a wokook. It i ok up ito tio o that you a put it dow ad pik it up lat, wokig though at you ow pa. p a. It i a lot to do at o, ad you do’t hav to do it all i o wk o v o moth. Do’t gt ovwhlmd. You a do it. Ad mm, hlp i alway lo y: you a gt hlp om ou wit, o you a d a utioal mdii dodo to to hlp you gu om o thi out i it i too muh o you. (s th appdix o ou.) My thid goal o thi ook i to o a mag o hop. My hat ig at th oppotuity to lt you kow that you do’t d to giv up ad giv i to a li o hoi uig ad mdiatio. I ily i ly wih ah o you a halig jouy a you mak th hag that I kow will hlp you l tt.
CHAPTER 1
AmmDsasBass
A HeALTHY iMMune SYSTeM You immu ytm ilud a goup o ll i you ody that pott you agait itio ad ill. Ti i why th immu ytm i o d to a a “amy” o ll. evy day wh you’ xpod to thig that ould au itio ad ill—uh a viu, atia, mold, paait, ad oig poti i ood—you immu ytm tak atio. o do o, it all upo may dit kid o oldi, ut to udtad autoimmu dia, w will ou o o attalio i patiula, alld lymphoyt. Lymphoyt a a typ o whit lood ll that i poil o pottig you om hamul oig lik itio. Howv, i thy a’t wokig ight, lymphoyt a th ll that au autoimmu dia. T a two kid o “oldi” that mak up th lymphoyt attalio. T t i kill ll, whih ditly attak aythig thy do’t ogiz ad whih thy piv a a ivad. I thik o thi dit attak a ll-to-ll omat. T oth kid o oldi i alld a b ll. T ll podu atiodi, whih a molul that ga o to aythig that you immu ytm thik i oig ad dagou. A th molul gt hold o th oig, you immu ytm iitiat a igg po that au a iammatoy atio. Wh thi happ, w ompoud a lad that attak th oig oig i od to kill k ill it ad la l a it out o you ody ody.. You You a thik o atiodi a ullt lad om th b ll to kill th t h ivad. ivad. both kid o oldi oldi o th immu ytm, atiody-poduig atiody-poduig b ll ad kill ll, tat a po that ult i iammatio thoughout th ody. Tough th po may gi omwhat ditly, th d ult you l i th am o th mot pat. T t ditio o a ompttt ad halthy immu ytm i o i whih th kill ll ompt ad th atiody-poduig b ll a i ala o that th immu po i alad, too. Dpdig o th ivad, omtim you atually l omthig i
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happig wh you immu ytm tak atio ad omtim you do’t. exampl o th oig ivad ilud atia ad viu. I you gt a iu o a itio, whih i aud y atia, you may xpi you immu ytm takig atio y havig a tud o ad pai i you a o iu aa. I you gt th u, whih i om a viu, you you might hav a high v v.. T ympto ymptom m a a th ult ult o o you you immu ytm tyig to ght th atia o viu. You might hav a tog atio ad th iammatio might lt i you mul o i you joit, lik athiti. All o th a ig that th immu ytm i wokig to d o th itio. I you immu ytm i tog, thi wa withi you hould top a a wk o two at th mot. O it jo i do, th immu ytm will lax ad go ak to it omal tat o wathig ad waitig o th xt od, ad th iammatio go away. I omo with a halthy immu ytm, thi i a good, omal po, ad w d th kill k ill ll ad atiodi to kp u halthy. T i mo tha o o typ o ll. T kill ad b ll a told what to do y th hlp ad th gulato, gulato, whih ith tu o o tu o th immu po. T dit typ o ll d to i ala o th immu ytm to tu o poply a it i ativatd ad th jo i do. Ti ala i th od ditio o a halthy immu ytm. Whil you immu ytm d to vigilat i od to guad agait itio ad toxi, it alo ha to vy aul ot to hut you ow tiu y mitakig you ow ll o th ivad. Duig thi alit dvlopmt, you immu ll hav to la th di tw omthig that i a atual pat o you ody, o “l,” ad
Three things that dene a healthy immune system: 1. Balance between between killer T cells and and antibody-producing antibody-producing B cells 2. Balance between T helpers and T regulators to turn on and o the immune system 3. The immune system’s system’s ability to dierentiat dierentiate e oreign invaders (such as viruses or bacteria) rom natural parts o your body (such as cells and tissues)
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a oig uta, o “ot l.” big al to mak thi dititio i alld tola. T thid ditio o a halthy immu ytm i o that attak oly ivad ad ot itl.
An iMMune SYSTeM Gone AWrY A autoimmu polm dvlop wh th immu ytm ail at all th o th ditio o halth. T ody gi to mak too may kill ll o too may atiodi (thi vai dpdig o th autoimmu dia ad will diud i dpth lat) ad th ail to tu o, o th immu atio do’t top. (T t two polm a alo i popl with athma ad allgi, au thy hav a ovativ immu po to uta alld allg. all g. symptom symptom uh a whzig ad ifig, ad v li-thatig togu wllig ad thoat tight, a aud y th immu po, ot th allg al lg itl.) but mot impotat o tho o you with autoimmu dia, th immu ll a attakig you ody’ ody’ ow tiu wh thy hould only only attakig outid ivad. Put all th polm togth ad th ult i iammatio ad damag to you ll ad oga.
WHAT Are AuToiMMune DiSeASeS? “Autoimmu” p p t t a category category o o at lat o hudd dia, ot o pi ill. Ti a ouig ad i poaly why may popl a’t amilia with autoimmu dia o a uu whih ill all ito thi atgoy. Futhmo, th am o th oditio, whih ilud Hahimoto’ thyoiditi, humatoid athiti, ytmi lupu ythmatou, sjög’ ydom, lia dia, ad multipl loi, amog oth, do’t hav th wod “autoimmu” i thm. Ti i ulik dia uh a th vaiou om o a, wh thi am otai th wod “a” ad th aa wh th maligat tumo() a oud. Fo xampl, at a i a tumo i th at, olo a a tumo i th olo, ad ki a a tumo o th ki. Without th wod “autoimmu” a pat o thi am, autoimmu oditio oud lik thy a dititly dit dia. Howv, that ould’t uth om th tuth. What a alo ouig i that th am o autoimmu oditio do’t tll you where th dia di a i loatd i th ody ody.. som autoimmu oditio a systemic systemic,, maig that th attak pad thoughout th ody to all tiu, a i lupu. Oth a oga pi, wh th
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attak ou i a pi aa o oga, lik Hahimoto’ Hahimoto’, whih ou i th thyoid. I ith a, th am i’t a hlpul idiato o wh th polm atually xit. Fo xampl, Hahimoto’ Hahimoto’ ad Gav’ dia dia a i th thyoid, multipl loi i i th ai ad pial od, vitiligo i i th ki, ad piiou amia i i th lood ll. Although th atd aa a dit, w ow kow that th udlyig u dlyig polm i all o th dia a vy imila. I at, th ou o t ah ha withd om lookig at th pi oga atd y th dia to dtmiig th udlyig mhaim o how th dia gi. Ti ida—that all o th oditio hav imila oigi—i itial to ou appoah o tatig ad vig thm. Mo tha o hudd dit autoimmu oditio hav imila haatiti. Ty a all iou hoi dia with a udlyig polm i th immu ytm. Aoth thig thy hav i ommo i iammatio, whih i iitatio ad wllig inside you ody o dy,, i ay pat iludig you ai. Iammatio Iam matio a au a wid ag o ymptom, iludig atigu, pu, mul o joit pai, adomial diomot iludig diaha, ad diulty otatig o “ai og.” O you may jut hav a vagu, aggig lig that omthig i’t ight, v i you doto a’t d aythig wog with you. by uig a utioal mdii app appoah oah ad ouig o th pimay au o th immu dyutio, dyut io, ah ha uovd may pottial tigg o th dia. (A tigg i aythig that that tat a uhalthy immu po.) It turns out that many autoimmune diseases are set o by similar things, such as gluten, heavy metals, toxins, inections, and stress. T mai di tw ah dia i that th immu ll tagt ad attak tiu i dit pat o th ody. etially, mot autoimmu oditio a mo alik tha thy a dit. Ad it tu out that xig th oudatioal ytm—whih a you dit, t homo, gut halth, halth , ad ody’ ody’ toxi load—will hal ha l th immu ytm ad hlp thm all. Ti i th volutioay appoah I dtail i Te Immune System Recovery Plan ad why th tatmt pogam it otai a t ad tagt all all autoimmu autoimmu dia.
WHAT cAuSeS AuToiMMune DiSeASe? T natioal Ititut o Halth timat that up to 23.5 millio Amia u om autoimmu dia ad that th pval i iig. With th um iaig vy ya, may xpt hav qutiod what au autoimmu autoimmu dia ad hav tudyig thi to d out.
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A a ult, th a may thought ad ida aout how you “gt” a autoimmu dia. H a th xplaatio with th mot vid hid thm.
PalTggoMd-DayDs Gluten Mod-day agiultual pati ilud omthig alld gti modiatio.. Ti ma that modiatio t hat th g i th d o op lik o, oy, ad what a altd i a laoatoy o that th plat a gow lag o it dia mo tivly. T ult o altig ou op thi way i that thy ow otai poti that a ot atual to th plat. Aimal tudi hav oud that th poti a xtmly diult o u to digt, whih au ymptom uh a: •
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Hatu rux Ga bloatig a atig
W hav alo vid that th poti au immu atio i th gut that a pomot th dvlopmt o autoimmuity. Autoimmuity ma that th ll o you immu ytm a damagd ad th mak a mitak ad attak you ow tiu. Glut i a poti oud i what, aly, kamut, ad plt, ad gti modiatio ha mad it tog ad mo otatd i th gai w at. Ti high otatio o glut i ou ood ha likd to th ia i ood allgi ov th lat w dad. Why? bau glut i a lativly w ompot o ou dit. Oigially, ou ato w hut-gath, o thy at aimal, ut, d, ad i, ath tha gai. T thy ttld dow to amig (oly t gatio ago) ad at aoally, otatig thi ood to at what wa availal duig that tim o ya. T t o thi i that you a otatly otatly vayig you dit, wha atig th am am thig all th tim ia you ik o dvlopig a allgi atio. Pod ood, whih a tho that hav altd y mauatu o thy o log look lik you jut gw thm, o hav all th ad may o th utit movd. Ti po wa atd to giv th ood a log hl li ad mak mo ood availal to mo popl, ut w ow kow that thi i ot a utitiou way to at. oday, popl
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who at th tadad Amia Amia dit at whit wh it ou at mot mal, o to th xluio o halthi, whol ood. Itad, you hould hoo whol ood, whih look lik thy did wh you pikd thm. T polm with glut i that it’ had to digt, ad wh a lot o lag pi gt ito th loodtam, th immu ytm go o high alt, ig th glut a a oig oig ad poduig atiodi to attak it. Uotuatly, wh th atiodi attak th glut, thy mitakly attak ou tiu a wll. Tis is called molecular mimicry and is one way gluten is believed to cause autoimmune disease. Molula mimiy i ot pi to glut. It a ou wh you immu ytm mitak you ow tiu o ay oig. T oth way ood a tigg iammatio ad autoimmu atio i alld immu-omplx dia. Uig glut a ou xampl, th atiodi id to th glut ad om a omplx that tavl aoud th ody o dy.. T a alld al ld immu omplx ad a a ommo, impotat way that you immu ytm dal with oig. You d immu omplx o th omal utioig o you immu ytm. nomally th immu ytm la th omplx out o th lood, ut i th a too may o thm, thy ttl i dit oga, auig loal iammatio, tiu damag, ad autoimmu atio. Ti a au woll, paiul joit ad i thought to o o th po o dvlopig humatoid humatoid athiti. Am I ayig that glut i th mai ao you hav a autoimmu dia? Fo om popl, it i; o oth, it i a ig pi o th puzzl. I lik to u th puzzl aalogy o thi au th a o val au o you autoimmu dia o immu dyutio, ad my appoah app oah i to add o pi at a tim. t im. I hav ho th ou tio o thi ook to dal with th iggt, mot ommo pi. O pat i ood, ut you mut alo add you t ytm, mak u you gut i halthy (do’t ogt that i pat two o th glut toy, au th glut might ot hav aud a polm i you ittial ai wa doig it jo), ad mak u you a’t ovloadd with toxi. O you add all th pi, you puzzl will omplt, ad wh you look at it you will a pitu o halth.
Fiber,, Fat, Fiber Fat , and Immune-Sys Immune-System-Supporting tem-Supporting Nutrients Oth dil id glut alo impat you immu ytm. A dit high i aimal-ad ood uh a daiy, gg, ad a pomot iammatio iammatio ad thow o th ala o good atia that liv i you
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digtiv tat. Fi ad vgtal a alo itial o thi atial al ala ad to ouih th liv o that it’ al to tivly mov toxi om you ody. (A you’ll ad lat i chapt 11, “suppotig You Liv,” toxi put you immu i mmu ytm y tm at ik, ik, too.) t oo.) Uotuatly, Uotuatly, may popl do’t at ough ad vgtal to ap th t. T a may utit that you mut ilud i you dit i od to hav a halthy immu ytm—om xampl ilud vitami D, vitami A, lium, zi, ad halthy at—ut th a o miig miig i th tadad Amia dit. Fo xampl, pod ood ll u up with ad at that au may polm id damag to th immu ll. W’ll talk aout thi i dtail i th xt hapt, “Uig Food a Mdii.”
PalTggchSssadHmimbala som popl do’t l td motioally, ut thy a kippig mal, ot lpig ough, o ovxiig. T havio tax you ody, auig it to t th t homo otiol ot iol om you adal glad. Oth popl may takig good a o thi odi, ut thy a axiou, woid, upt, o dpd, o thy hav v, ogoig motioal tauma. T havio au th am otiol po om you adal glad. T adal glad a mall odul that it o top o you kidy ad mak all o you t homo. homo. I wat to la that ot all t i ad. Fo xampl, i a mgy ituatio, you adal glad la otiol ad adali, whih povid you with th gy to mov quikly to gt hlp. O o a impotat talk, it giv you th gy to ou ad thik. but chronic t ma you lvl o otiol a otatly lvatd, omthig that a damag you immu ytm ad pvt it om halig. choi t a alo lad to what i alld adal atigu, whih i wh you adal glad gt xhautd ad do’t podu th homo quid to kp you ody uig poply, iludig adali, DHeA, ad ttoto. Adal atigu ult i: •
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Uxplaid xhautio Flig lik you a’t gt up i th moig v a a good ight’ lp A ut o gy tw 4:00 ad 6:00 p.m. Flig ovwhlmd cavig o wt o alty ood
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Low lood pu pu Low lood uga Iitaility
Adrenal atigue (also known as adrenal exhaustion and adrenal burnout) is associated with infammation and autoimmune disease, which is why it’s so important to understand and better manage the stress in your lie. st a alo hav a gativ t o th lvl o good atia i you digtiv tat, whih itl a au autoimmu dia. st homo may alo th polm i you a atigud all th tim, gt ik o, hav dvlopd athiti, xpi igula piod, a goig though a diult mopau, o hav toul loig wight. Ti will diud i dtail i chapt 5, “Udtadi “Udtadigg th st coc otio.”
PalTggAimbalaGdBaaYG You immu ll, pially th t h kill ll ad b ll, a th mot tal to th autoimmu polm. It i wh th ll do’t wok ight that th at th ody tat t at attakig itl it l ad do’t do’t top. I od to hlp th ll ll utio u tio tt, it’ impotat to udtad udt ad how thy dvlop. dvl op. Wh you a a adult, immu ll a mad i you o maow ad th migat to th thymu (a mall oga ud you ato), you lymph od, ad a aa alld th t h gut-aoiatd lymphoid lymphoid tiu (GAL, jut ud th ua o th ittial liig). T thymu wa vy ativ wh you w i you moth’ moth’ wom, ad wh you w o, it wa th mai hom o you immu ll. ll . A you ag, it till hlp th ll matu matu ad dvlop, ut it om l ad l ativ. T liig o you itti hould otai good atia (alo alld oa) that a itial o hlpig th immu ll matu poply au thy itat with th ll i you GAL. Wh th good atia a’t ouihig, th immu ytm i uptil to dyutio. sval thig a impat you good atia. A I jut mtiod, o i t. Aoth i that w a a oity livig o what I all th v A’: ataid, atiioti, alohol, Advil, ad aimal ood. T thig (alog with itio ad oth mdiatio) alt th ial atia i you itti ad damag th ai o th ittial wall o that ood lak ito th GAL aa aa low th ittial liig, ad th ito you loodtam. Wh thi happ, th immu ytm ogiz
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th ood patil i you lood a oig ivad ad dvlop atiodi to attak that ood. A a ult, you may hav a atio to a ood that you’v atig all you li. T oth impotat ol o th ial oa i that thy hlp th kill ll i th gut liig dvlop ad la th di tw a oig uta (i.., a itio o atia) ad you ody’ ow tiu. Ti i why halig you gut y makig u th ial ial atia ad ittial liig a th halthit thy a i o o th udamtal way to kp you immu ytm halthy. W’ll talk mo aout thi i chapt 8, “Halig You Gut,” ut it’ impotat to kow that a healthy gut is critical or a balanced and well-unctioning immune and ha ha th pottial to system. It a hlp pvt autoimmu dia and tat you ymptom ad hal you immu ytm.
PalTggTxs A toxi i ay viomtal hmial, havy mtal, o oth ompoud that i oig to th ody ad au a hamul atio o ay kid. Ti a alo ilud mold au it ommoly giv o dagou toxi. eviomtal evio mtal xpou to toxi, whih w hih a damag oth th immu ytm ad oth ll i you ody ad lad to autoimmu dia, i omthig w’ xpiig today at updtd lvl. I at, th ct o Dia cotol’ (cDc) Fouth natioal rpot o Huma expou to eviomtal chmial ttd 212 hmial ad oud that all all o o thm w i th lood ad ui o mot Amia. 1 Ti i ot upiig, i w’ outily xpod to toxi though ood, ptiid, goudwat, idutial wat, ad idutial hmial. With pt to autoimmu dia, w a patiulaly od with ay toxi that hag th hmial tutu o ou DnA, it it gti matial alld rnA, ad th poti i you ll au thi a timulat a immu po i you ody. I oth wod, th toxi hag you tiu tutu, ladig you ody to you ow tiu a a oig uta ad th attak it. T mot wll-tudid toxi i latio to autoimmu dia i muy. (O th 212 toxi oud i th cDc’ pot, thi wa i th top ix.) Muy xpou om om ilv-olod dtal amalgam llig ud o aviti. It i alo lad ito th atmoph a a y-podut o uig oal o wood o ul ad th iiatio o muy-otaiig matial. bau thi ha goig o o may dad, th muy om th ai ha ttld ito ou oil, iv, ad
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oa. A a ult, u lt, it’ it’ i may o th h w at, at, uh a wodh, tua, tipd a, ad kig makl. (si muy otat otat up th ood hai, lag h who at th littl h td to otai th hight lvl.) studi hav likd muy to Hahimoto’ thyoiditi, Gav’ dia, lupu, ad Ms. It appa that muy i o o th toxi that ditly damag you tiu, makig thm look oig to th immu ytm. by ow you kow that th immu ytm attak aythig that it do’t ogiz. Tis is why a crucial part o Te o Te Immune System Recovery Plan is to assess your potential toxin exposure and then take steps to remove as many toxins as possible p ossible rom your diet and environment. Ti i what w will do i Pat IV, “suppotig You Liv.” Aoth uial iu with toxi xpou i that wh th ody otai too may toxi, th liv, ou mai dtoxiyig oga, gt tid tyig to la thm out. Tik o it a liv atigu. T liv ha may dtoxiatio pathway, zym ytm that a poil o movig toxi. eah o qui pi utit, ad i th a too may toxi ad ot ough utit, th liv gt dpltd ad toxi aumulat. T liv i alo poil o hlpig po th homo you ody at atually. Wh th liv gt xhautd du to high lvl o toxi i th ody ody,, it alo tuggl with it daily jo o poig ou ow homo ad th hmial atually atd y th ody. etog i patiula i mtaolizd though th liv, whih otai pi zym ytm that d to wokig wll i od o thi homo to pod ad poply movd om th ody. Howv, i th liv i td, th tog uild up ad th ody atually podu more toxi tog, whih a au DnA damag ad pomot a immu atio. I at, toxi tog a thought to play a impotat ol i oth lupu ad humatoid athiti. A you will wi ll la i Pat IV, “suppotig You Liv,” pi ood ad upplmt a hlp homo, hmial, ad toxi ow though th liv mo itly ad tivly.
PalTggis T i a lot o litatu likig viu to autoimmu dia, ad I will diu th pottial likag lat. but th olutio i ot to lam th viu. All o u hav viu i ou odi; it’ ou immu ytm’ jo to kp thm i miio miio. . by thi t hi I ma that t hat th viu a uppod to diald o that thy do’t mak u ik. Howv, i a viu i ativ, th immu ytm mai o a hightd tat o alt ad
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thi ma ogoig iammatio i th ody. Ti i a polm. symptom o thi td to vy gal, uh a lig puy, woll, ti, ad tid ad/o havig diulty thikig ad mmig thig. Fo xampl, thi i vy ommo with th viu that au moouloi (moo), whih ha impliatd i autoimmu dia. calld th epti-ba viu, it tay i you ody ody ov, ov, omtim udttd, at oth tim auig polm. I hav may patit tll m that thy v lt quit th am a havig moo ad wh I tt thi lood o th epti-ba viu, I o d that it i till til l ativ. T olutio to thi i to udtad why why you you immu ytm ha aild to upp th viu. Ti i xatly what w will do i Te Immune System Recovery Plan a w wok to tgth ad ala th udamtal oudatio o a halthy immu ytm (dit, t, gut halth, ad duig you toxi load). A a ult, you immu ytm a dial th viu vi u ad pvt thm om pittly timulatig it.
HoW To uSe THiS Book: Your GooD HeAL HeALTH TH DreAM TeAM A I mtiod ali, autoimmu dia hav om th mot pvalt om o hoi h oi ill i ou outy out y. Yt Yt th till t ill ha’t ha’t ough atttio atttio paid to thm. I thik thi i why popl viw th oditio a dit ill. T doiologit th patit with Hahimoto’ thyoiditi ad Gav’ dia; th humatologit th patit with humatoid athiti ad lupu; th gatot gatotologit ologit tho diagod with lia dia; dia ; ad th uologit th multipl loi patit. A a ult, th i ot o uid appoah, whih I liv ha lowd th advamt o a tt udtadig o autoimmu dia ad mo appopiat tatmt o thi oot au. Aoth iu i that mot ovtioal tatmt o th oditio ou o otollig ymptom with mdiatio that wok y huttig o th ody’ ody’ immu po. po . Ti i’t i’t alway tiv t iv ad i o aompaid y iou id t. Fo xampl, toid uh a pdio a au iomia, wight gai, iad lood pu, mul pai, ad dpio. dpio. Oth dug a ud to dial th immu ytm ad a hav mo v t o th digtiv tat uh a aua ad vomitig, ut alo v, mul pai, amia, ad uig itio. Ty alo a damag th liv, lug, ad kidy. bau om o th dug a tay i you ody o up to two ya aer aer you’v you’v ihd uig thm, thy a vy dagou i you gt pgat withi
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that tim am. Ti i a itial iu i autoimmu dia at wom 75 pt o th tim. Ti tatiti t atiti ha ld may ah to look at th ol o x homo i th dvlopmt o autoimmu dia. I will ha mo aout thi topi wh w talk aout t ad t homo, dtox, ad tog mtaolim though th liv, ad wh w talk aout lupu i th lat hapt. but th mot impotat polm with th dug du g i that thy a tatig oly th symptoms o you autoimmu oditio, ot th cause o it. Ty do’t xplai why why you you immu ytm i ot wokig poply i th t pla, ad i you do’t gu thi out, you a oly maag you ymptom ath tha u you oditio ompltly. I a taditioal doto upt that you hav a autoimmu dia, th t lood tt h o h will do i what’ alld a ati-ula atiody (AnA) tt. Ti do’t look o ay o pi autoimmu dia ut i a gal ig tt o ytmi autoimmu dia uh a lupu. You doto might alo do om tt o dit ogapi dia, uh a Hahimoto’ o Gav’ dia. A I mtiod ali, i a halthy immu ytm atiodi tagt ad attak oig that a au itio ad ill. Wh a autoimmu oditio dvlop, th atiodi tagt you own tiu, ad o th t atiody to how up i laoatoy tt tt i th ati-ula atiody. I th AnA tt i poitiv, th doto do to do pi tt o lupu, humatoid humatoid athiti, sjög’ ydom, lodma, mixd otiv tiu dia, polymyoiti, o dmatomyoiti. I all th tt a gativ ad you jut hav a poitiv AnA, you’ ot diagod with a autoimmu dia. At lat ot yt. T ovtioal mdial appoah i to wath ad wait to i you ymptom gt wo ad you tt poitiv vtually.. Ti i all with th ally t h xptatio that omday you will will dvlop dvlop o o th pi dia. Ti wath, wait, ad do othig appoah i agait all o th piipl o pvtiv mdii ad utioal mdii au th i o muh that w a do to preven to prevent t th th dvlopmt o ull-low autoimmu dia. It tu out that you a hav a poitiv AnA tt o may ya o you dvlop ay o th atual dia o o you hav ay ymptom. Fo xampl, you a hav ati-thyoid atiodi o may ya o you oti a polm with you thyoid utio. You a hav a immu atio to glut o may ya o you how ay ig o lia dia uh a damag to th mall itti. My goal, ad that o pvtiv pvtiv mdii ad utioal u tioal mdii, i to ath
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th atiodi aly ad th x th immu ytm y diovig why it i dyutioal. Ti way w a quit dow th kill ll ad th atiodi, pvtig thm om doig tiu damag ad tuig ito a ull-low dia. by ow you udtad that atiodi ad kill ll a good wh thy a mad agait ad thig uh a hamul atia, viu, o a ll. but w do’t wat to hav atiodi o kill ll attakig ou omal, halthy tiu au it will gi a aad o damag, iammatio, ad vtually impaid utio. Fo xampl, i popl with humatoid athiti, th atiodi that dpoit i th joit au damag that dom th joit itl, auig pai ad impaid utio. I lupu, atiodi a attak th ll liig th lood vl, auig damag to th lood upply to th oga wh thi i happig. (Popl with lupu o gt kidy damag thi way.) Ti i why it i itial to d th atiodi aly, beore th i damag i joit, lood vl, o aywh l i th ody o dy.. rah pov that thi a do, ad I kow it o tai au it i what I do daily with my patit i my o. It i how I ud myl, too. Ti ook will how you you how.
HoPe iS Here My goal i witig Te Immune System Recovery Plan i to ig a mag o hop. You do’t hav to it ad wait to gt a dia that i p vtal. Ad you do do’t ’t hav to it ad wath you dia gt wo, thikig that th i othig that a do to v it. I you’v alady diagod, it i ot too lat. You do hav optio id takig piptio dug o th t o you li. Ad my goal i to hlp you that. by ollowig th tp i thi ook, you a l halthy agai (y, halthy!) ad v you dia. but lt m t mak o thig vy la. I am ot ati-mdiatio. I you a havig a a o you dia, maig you ymptom gt wo ad you’ i til pai ad l vy ik, ovtioal mdiatio a vy hlpul ad ay. ay. but o thi ii ha pad, you ou hould hi to guig out th oot au o th immu dyutio ad xig that. Alo, utioal mdii i ot a altativ appoah. app oah. I am a mdial doto ad I wok with my patit ad taditioal phyiia, v whil my patit a takig mdiatio. I wok to x th ouda oudatio tio o thi immu ytm o that all th ymptom ymptom
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ad atiodi diappa. Wh thy a ady, thi doto ad I did togth how to go aout tapig o th patit’ mdiatio. I thi ook, I will pt my ou tatmt pogam o you to do o you ow. I you’ takig mdiatio o you autoimmu dia, you a till do th pogam. Howv, i you’ od, talk to you doto aout th pla. Kp i mid that may o th uggtio ad tatmt a lityl hag ad do’t qui you to do aythig that would mak you o you doto uomotal. but alo kow that jut au you doto i’t amilia with om o th thig I’m uggtig, that do’t ma thy’ dagou o ad o you. It may imply ma that you doto ha’t ad th tudi ad/ o lad aout thi appoah. Do’t diouagd y thi. I’v oud that may o th doto i my ommuity who o w kptial o utioal ut ioal mdii a ow ag to d m patit ad wok with m to hlp thm. Why? bau thy that thi appoah o ig t with vy littl ik. I am paioat aout thi au it i tuly a logial appoah to tatig th au o th autoimmu polm. Ti go yod imply tatig ymptom. It ma that th i al hop au th i omthig omthig w a do to hlp you tat, v, ad p vt dia.
THE MOST COMMON AUTOIMMUNE DISEASES The most common autoimmune diseases that I see in my medical practice are Graves’ disease, Hashimoto’s disease (also known as Hashimoto’s thyroiditis), lupus (more ormally known as systemic lupus erythematosus), multiple sclerosis (MS), rheumatoid arthritis (RA), Sjögren’s syndrome, and celiac disease. I also see other autoimmune diseases, including glomerulonephritis (a kidney condition), type 1 diabetes, pernicious anemia (destruction o red blood cells), and vitiligo (a skin condition). For our purposes, I am going to ocus on the seven diseases I see most. Here is inormation on these common autoimmune diseases, their symptoms and important tests to get i you suspect you have one. But remember, it doesn’t really matter i your condition is on my list or not. You still need to x your oundational systems i you have an autoimmune disease.
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CELIAC DISEASE This is a disease caused by an allergy to gluten and is marked by destruction o the microscopic, nger-like protrusions called villi that line the small intestine. It may take many years o gluten exposure beore the villi are damaged and a laboratory conrms that you have celiac disease, but in the meantime the gluten can cause other digestive and autoimmune issues beore it is diagnosed. Celiac disease has become the most well-known autoimmune disease because so many people have developed sensitivities to gluten.
SymptomsofCeliacDisease Gluten can cause autoimmune diseases in other organs in addition to the gut, so there are a wide range o symptoms, rom numbness and tingling in the extremities to atigue rom low thyroid unction. Some common symptoms include: •
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Arthritis Generalized brain og Generalized atigue Digestive issues such as diarrhea, gas and bloating aer eating, and heartburn Anemia
TeststoRequestfromYourDoctororHealthCareProfessional There is a lot o conusion about how to diagnose celiac disease. Gastroenterologists will give you this diagnosis only aer a biopsy showing damage to the villi o the small intestine. This is very restrictive, since you might have what’s called silent celiac disease or decades beore this test is positive. Instead, ask your doctor or tests or anti-gliadin antibodies and anti-deamidated gliadin antibodies. These tests are more sensitive in picking up gluten allergies and can be positive or many years beore there is any damage to your small intestine. I these are positive, it’s a sign that an autoimmune attack is taking place somewhere in your body. In that case, you should assume you have very early celiac disease that hasn’t aected your intestines yet but is doing plenty o damage in your body, perhaps showing up as Hashimoto’s thyroiditis, Graves’ disease, multiple sclerosis, or another autoimmune disease.
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And just to add to the conusion, even i all the above tests are negative, you might still be sensitive to gluten. That’s because these tests were designed to pick up celiac disease only and gluten can cause other autoimmune diseases dis eases as well. Thereore, The reore, i you have any autoimmune disease—not necessarily celiac disease—it is good to do the tests above, but i they are negative, you should still remove gluten rom your diet, based on research showing a connection between gluten and many other autoimmune diseases.
GRAVES’ DISEASE Graves’ disease happens when your body makes antibodies that stimulate your thyroid gland, causing it to secrete high levels o the hormone thyroxine (also known as T4). This condition is called hyperthyroidism.
SymptomsofGraves’Disease •
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Weight loss Rapid pulse Protruding eyes Insomnia Feeling too warm Restlessness Diarrhea Irritability Heart palpitations
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Thyroid-stimulating hormone (TSH) Thyroid-stimulating Free T4 Free T3 Thyroid-stimulating Thyroid -stimulating immunoglobulins (TSI) TSH receptor antibody
Here is the pattern o test results you would expect i you have Graves’: •
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TSH is low, typically <0.5 mIu/L, oen lower or undetectable. Free T4 is elevated, usually over 2.5 ng/dl.
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Free T3 might be normal but is usually over 4.0 pg/ml. Either the TSI or the TSH receptor antibody will be positive. I they are both normal, then you don’t have Graves’ disease.
The above pattern is what the numbers would look like in a typical, classic case o Graves’ disease. However, sometimes only one o the numbers looks out o range, such as a high ree T4 with a normal TSH. This is a sign that you might have caught the problem early, and it is the perect time to go through the steps in this book and reverse the problem beore the disease starts.
HASHIMOTO’S THYROIDITIS Also called chronic autoimmune thyroiditis, this is the most common autoimmune disease. Here, the immune cells invade the thyroid. In early Hashimoto’s thyroiditis, the thyroid still unctions pretty well, so i your doctor is checking only your TSH and hasn’t measured the antibodies, you might miss the early stages o this condition. This is unortunate because early on is the perect opportunity to reverse the antibodies and prevent thyroid damage. I the immune attack goes on or too long, the thyroid might become permanently damaged, requiring lielong hormone replacement.
SymptomsofHashimoto’sThyroiditis •
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Enlarged thyroid (goiter) I your thyroid is actively inamed, some people experien experience ce a sore throat. Fatigue Hair loss Weight gain
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TSH Free T4 Free T3 Anti-thyroglobulin and anti-thyroid perox peroxidase idase antibodies
Here is the pattern o test results you would expect i you have Hashimoto’s:
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One o your antibody levels will be elevated, either thyroid peroxidase perox idase antibodies or anti-thyroglobulin antibodies. I these are normal, you don’t have Hashimoto’s. TSH, ree T4, and ree T3: I these levels are normal, you are not hypothyroid. In early Hashimoto’s, you can have the autoimmune disease and the thyroid is still making adequate amounts o hormones. This is the perect time to ollow the steps in this book, because you have caught the problem early while it is reversible and can prevent damage to your thyroid gland. Here are my suggested normal values or the hormones or screening purposes: TSH: <3.0 mIu/L FT4: >1.0 ng/dl ng/dl FT3: >2.6 pg/ml I the TSH is over 3.0, 3.0, or i the ree T4 is under 1.0 and the ree T3 is under 2.6, your thyroid might be starting to show signs o damage rom the autoimmune disease. You can discuss with your doctor whether it is a good idea to take a prescription thyroid hormone replacement. I will talk more about treating Hashimoto’s in Chapter 14, “Inections and Specic Autoimmune Conditions.” •
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LUPUS Lupus, which is more ormally known as systemic lupus erythematosus, involves more tissues in the body than other autoimmune diseases because it is a condition in which the body creates antibodies against the DNA o the cells. As a result, you can end up with disease all over your body and can have ever, joint, and muscle pain. Keep in mind that the symptoms come and go, because the disease can cycle through being in remission and being active. Unortunately, many lupus patients get very sick, oen dying rom the involvement o the small blood vessels, which gives them disease in all their organs, including the kidneys and heart. Lupus aects more women than men, especially those in their twenties and thirties, leading researchers to believe that estrogen plays a role in causing or triggering the disease. I will explain more about this in Chapter 11, “Supporting Your Liver.”
Amm Dsas Bass
SymptomsofLupus •
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Fatigue Muscle pain and weakness Fever when the disease is active Symptoms Sympt oms specic to the organ involved, involved, such as joint pain, muscle pain, and difculty breathing Buttery rash over the cheeks and nose that appears aer sun exposure Hair loss (but not baldness) Oral or nasal ulcers that are not painul Cold- or emotion-induced color changes o the ngers or eet
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Anti-nuclear antibodies Anti-phospholipid antibodies Antibodies to double-str double-stranded anded DNA Anti-Smith (Sm) antibodies
The ANA test is the rst screening test or lupus. As I have explained, a positive test doesn’t mean you have lupus unless one o the other three tests is positiv positive e as well.
MULTIPLE SCLEROSIS (MS) Myelin is the protective coating on the outside o all the nerves in your body. In those who have MS, the myelin in the brain and spinal cord is damaged. This damage is called sclerosis. MS primarily aects women who are o northern European descent and who are o childbearing age. The most common rst symptom is an episode o central nervous system dysunction, such as optic neuritis. This is eye pain that gets worse when you move your eye in any direction. Sometimes symptoms go away on their own. Each time they come back, it’s called an episode or exacerbation o symptoms.
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Eye pain Numbness, tingling, or pins-and-needles sensation anywhere in the body that doesn’t go away aer two weeks
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Swelling o the limbs or trunk Intense itching sensation, especially in the neck area
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There are no antibody tests or MS. Instead, it’s diagnosed when lesions in the brain or spinal cord are seen on an MRI. It’ It’ss important to note that the diagnosis is made only aer having neurological symptoms symptoms twice or a second episode that shows a second lesion in the brain or spinal cord. One episode that resolves and never comes back is not considered MS.
RHEUMATOID ARTHRITIS (RA) I you have arthritis, it’s oen difcult to tell the dierence between symptoms o RA and common osteoarthritis pain and swelling that can occur with aging or aer injury. Rheumatoid arthritis occurs when your immune cells attack your joints, causing tissue damage, inammation, and pain. It is a very specic orm o arthritis, and sometimes the only way to know which kind o arthritis you have is to do the blood tests listed below.
SymptomsofRA •
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Muscle pain Fatigue Low-grade ever Weight loss Depression Morning stiness that lasts at least one hour or at least six weeks Swelling o three or more joints or at least six weeks Swelling o wrist or ngers or at least six weeks Symmetric joint swelling Nodules or bumps under the skin and over an aected joint
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Hand X-ray Blood tests or ANA, rheumatoid actor (RF), and anticitrullinated peptide/protein antibodies (anti-CCP) Blood tests or inammation: ESR (erythrocyte sedimentation rate) and high-sensitivity C-reactive protein (sometimes called a Cardio CRP)
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It is good to get all the above blood tests because they will determine i you have rheumatoid arthritis. It is possible to have a positive ANA with all the rest o the tests being negative. In that case, you don’t have RA. The opposite can also be true: you can have RA because you have a positive RF or anti-CCP but have a normal ANA. The ESR and the Cardio CRP are indicators o how much inammation might be happening at the moment, helping to monitor ares in the disease.
SjöGREN’S SYNDROME Sjögren’s syndrome, which can occur on its own or in conjunction with RA, is an attack on the mucus-secreting glands that causes a reduction in secretions. It is oen elt rst in the salivary glands, which are in the mouth, and lacrimal glands, glands , those that secrete tears. Ninety percent o patients are emale.
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Dry mouth and dry eyes Dryness in the vagina, skin, lungs, sinuses, and digestive tract Fatigue Joint pain Muscle pain Cognitive dysunction
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ANA, anti-SSA, and anti-SSB antibodies
Elevated levels o anti-SSA or anti-SSB anti- SSB are diagnostic or Sjögren’s.
SYMPTOMS TO TAkE SERIOUSLY Below is a checklist o symptoms or common autoimmune diseases. I you have any o the boldaced ones, you should go to your doctor and
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ask to be tested or the disease or diseases I have mentioned. The other (not boldaced) symptoms in the ollowing lists are nonspecic, meaning many other things besides autoimmune diseases can cause them. I you have our or more o the nonspecic symptoms and they are all consistent with one condition, then t hen you should have the test suggested or an ANA screening. For example, i all your symptoms t with lupus but you don’t have any o the specifc lupus symptoms, you should still get tested. This is very important because with many o these conditions, the symptoms don’t make a diagnosis; the lab tests do. I have also included several additional autoimmune diseases in the checklist below because these are airly easy to identiy and get tested or.
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Fatigue: all autoimmune diseases General discomort, uneasiness, or eeling ill: all autoimmune diseases Insomnia: Graves’
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I you are having evers but you don’t have a virus or inection, or i you eel hot h ot all the time: lupus, Graves’, Graves’, celiac disease, Sjögren’s I you eel hot when others are cold: Graves’ thyroiditis with an overunctioning thyroid gland I you eel cold when others are hot: Hashimoto’s thyroiditis with a low-unctioning thyroid gland
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Hair loss, usually in patches or circles: alopecia areata (this is conrmed by examination; there is no blood test) Loss o all the hair h air on your body: alopecia universalis (this is conrmed by examination; there is no blood test) Thinning o your hair or general hair loss: celiac, lupus, Hashimoto’s with low-unctioning thyroid gland
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Dry skin: Hashimoto’s thyroiditis Bruising easily: celiac
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Itchy skin: celiac Rash over your cheeks and bridge o the nose (buttery rash), usually red with some bumpy texture texture (but no pimples), that gets worse in sunlight: very specic or lupus Skin sensitivity to the sun: lupus General rash anywhere on the body: lupus Fingers that change color when cold: Raynaud’s phenomenon, lupus Nodules or bumps under the skin, usually on your hands or eet: rheumatoid arthritis Skin thickening: scleroderma Loss o skin pigmentation in blotchy patterns anywhere on the body: vitiligo (this is conrmed by examination; there is no no blood test)
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Vision changes: lupus, MS Dry,, itching eyes or eeling like there is something in the eyes: Dry Sjögren’s, Sjögren’ s, rheumatoid arthritis Double vision, eye discomort, uncontrollable unco ntrollable eye movements: movements: MS
Throat,Nec,Voice,andMouth •
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Swollen glands (lymph nodes): lupus, Sjögren’s Enlarged neck rom rom an enlarged thyroid: Hashimoto’s Hashimoto’s Mouth sores or ulcers: lupus, celiac, Sjögren’s Difculty swallowing or speaking: Sjögren’s, MS Loss o sense o taste: Sjögren’s Hoarseness: Sjögren’s Dry mouth: Sjögren’ Sjögren’s, s, RA Excessive Excessiv e thirst: type 1 diabetes
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Joint pain or joint swelling: RA, Sjögren’ Sjögren’ss Morning joint stiness lasting more than one hour: RA Pain and tenderness throughout the body: Sjögren’s, lupus Muscle weakness: Hashimoto’s, Graves’, MS Muscle cramps and joint pain: celiac Muscle spasms and twitching: MS
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weightChanges •
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Unexplained weight loss: Graves’, celiac, lupus, type 1 diabetes Unexplained weight gain: Hashimoto’s, gluten sensitivity (not celiac), type 1 diabetes
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Constipation: Hashimoto’s, celiac, MS Abdominal pain: celiac, lupus Bloating, gas, or indigestion: celiac Diarrhea, either constant or on and of: celiac Nausea and vomiting: celiac, lupus, Graves’ Stools that oat and are oul-smelling, bloody, or “atty”: malabsorption malabsorp tion rom celiac
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Difculty concentrating: Hashimoto’s, MS, Graves’ Depression: celiac, MS Irritability or anxiety: Graves’, Hashimoto’s
BalanceandNeurologicalS BalanceandN eurologicalSymptoms ymptoms •
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Numbness and/or and/or tingling in the th e extremities: extremities: lupus, MS, celiac, type 1 diabetes Headaches: lupus Seizures: lupus, celiac Problems walking, loss o balance, coordination: MS Tremor: MS, Graves’ Dizziness, vertigo: MS
T puli’ itt i autoimmu dia i xtiv, ad popl a lamoig o mo iomatio. Poo o thi i th at that autoimmu dia ad diod a th mot popula halth topi qutd y all to th Dpatmt o Halth ad Huma svi’ natioal Wom’ Halth Iomatio ct.
Recipe Index
Breakfast
Almond Blueberry Mufns Belly-licious Smoothie Blueberry Spinach Smoothie Green Detox Soup Homemade Granola Pesto Scrambled Eggs Warm Quinoa Cereal with Fruit and Nuts Weekend Frittata
93 217 282 282 217 160 95 160
Snacks
Almond Butte Butterr Granola Bars Almond Blueberry Mufns Belly-licious Smoothie Green Detox Soup
94 93 217 282
Entrees
Black Bean Burger Burgerss Buddha Rice Bowl with Tahini Dressing Chicken Stued with Red Peppers, Pine Nuts, and Spinach Coconut Chicken with Almond Lime Sauce Cod with Puttanesca Sauce Maple Teriyaki Salmon Mediterranean Mediterra nean Chickpea Pat Patties ties with Red Bell Pep Pepper per Sauce Mediterranean Mediterra nean Herb-Crusted Salmon Mung Bean Kitchari Mushroom Tempeh “Scaloppine” North Arican Red Lentil Soup Quinoaa Pasta with Peas, Arugul Quino Arugula, a, and Sun-Drie Sun-Dried d Tomatoes Turkey Burgers with Caramelize Caramelized d Onions
289 97 222 169 168 288 166 99 219 165 283 98 221
rp idx
349
Salad dishes Aia soa noodl salad chikpa salad Kal salad with ahii Dig Lmoy Kal ad Avoado salad Oag, Fl, ad Gold bt salad spiy blak ba Quioa salad spoutd spo utd Fh Ltil salad
96 285 286 163 221 162 218
Side dishes Aia slaw Havt Wild ri ciamo Mahd swt Potato camd spiah Galiky G i coout Oil Gig bok choy sautéd Whit ba ad Galiky G Whippd cauliow Wild Muh Muhoom oom Quioa
290 287 164 100 220 163 284 223 101
Dessert bluy Paai Paaitt with cahw cam choolat Avoado Puddig choolat chip Oatmal cooki
224 170 102
Index
Pages in italics refer to illustrations abdomen, 146 alcohol, 8, 18, 149, 263 detox elimination diet and, 269–71 cramping in, 180, 185–86, 195 fat and, 78, 110–11 and eating for immune health, 78–79 gut and, 179, 183, 188, 193, 207 gut and, 180, 183–86, 195, 197, 213 pains in, 14, 34, 48, 183–84, 213 stress and, 124, 133 Sjögren’s syndrome and, 183–84 aldosterone, 112 allergens, ix–xi, 13 acne, 154, 198, 263 acupuncture, 136, 148, 151, 156 allergies, 9, 13, 181, 208, 318 see also food allergies adaptive immune system, 117, 176 adaptogens, 155 almonds, 42, 55, 82, 154, 158, 207 Addison’’s disease, Addison dis ease, 112, 183 detox elimination diet and, 270–72 food sensitivities and, 66, 68–69 adrenal fatigue, 169 diagnoses of, 121–22 recipes and, 93–95, 102, 169–70 perimenopause and, 60–61 aloe, 206 alpha lipoic acid (ALA), 251, 255–56, 277, self-assessment and, 132–33, 137–42 stress and, 17–18, 113–14, 119–21, 301, 330 aluminum, 232, 240, 242–43 132–33, 137–42 treatment program for, 142–43, removal from body of, 255–56, 261 156–57 amino acids, 182 detox and, 248, 250, 276–78 adrenal glands, 17, 108 DHEA and, 112–13, 122–23, 156, 312 patterns of, 43–44, 50 recipes and, 215, 221–22 herbs for, 155–56, 330 lupus and, 305–6 androgens, 122, 312–13 perimenopause and, 60–61 anemia, 21, 48, 182, 197 celiac disease and, 25, 47 recipes and, 158, 160–61, 165, 169 stress and, 106–10, 112–14, 119–23, pernicious, 14, 24, 183, 241 129–30, 132–33, 137–43, 148–49, antacids, 8, 18, 44 gut and, 179, 182–83, 186, 196, 199, 152, 158, 330 supplements for, 143, 155–57, 330 206–8 antibiotics, 8, 18, 43, 169, 238 treatment program for, 143, 148–49, 152, 154–57 gut and, 178–79, 186, 188, 192, 196, adrenaline, 17, 107, 124, 155 204, 211 MS and, 298–300 recipes and, 158, 170 stress and, 106–9, 114–20, 129, 137, antibodies, xi, 7, 13, 22–31, 57, 86, 115–20, 117, 242 140 aerobic exercise, 153 celiac disease and, 25, 51, 72, 275, 307 agar, 207, 224 gluten and, 16, 62, 294–95 Graves’ disease and, 26–27, 61, 120, agriculture, 15, 46, 67 toxins and, 229, 236, 238, 244–45 241, 314–16
idx gut ad, 19, 176, 179, 183, 189–90, 194–95, 295 Hahimoto’ thyoiditi ad, 4, 27–28, 194, 241, 314, 317 halthy immu ytm ad, 11–12, 22 immuoglouli (Ig), (Ig), 26–27, 45, 61, 124, 179, 300, 302, 305, 316, 332 lupu ad, 23, 28–29, 193, 240, 302–5, 312–13 Ms ad, 30, 297–300 rA ad, 23, 30–31, 37, 193, 308–13 sjög’ ydom ad, 31, 184–85, 193, 309, 312–14 t ad, 118–19 toxi ad, ad, 240–41, 244–45, 318 see also b lymphoyt atiody ll; specifc cells ati-itulliatd ati-itullia td pptid/po pptid/poti ti atiodi (ati-ccP), (ati-ccP), 30–31, 193, 308–9 ati-damidatd ati-da midatd gliadi atiodi (ADGA), 25, 51, 72, 275, 307 atig-atiody omplx, 45 atig, 57, 124, 175–76, 310 gut ad, 176, 189–90 ati-gliadi atiodi atiodi (AGA), 25, 51, 72, 275, 307 ati-iammatoy ati-ia mmatoy dit, 53–54, 59, 88, 268, 302 ad atig o immu halth, 72–82 ood itiviti ad, 72–73 good at i, 72, 81–82 a low i uga, 74–81 aiow o vgtal i, 73–74 ati-ula atiodi (AnA), 22, 194 lupu ad, 29, 193, 240, 302–3, 309 rA ad, 30–31, 193, 308–9 sjög’ ydom ad, 31, 193, 309, 312, 314 toxi ad, ad, 240, 244–45 atioxidat, atiox idat, 57–58, 268, 319 dtox ad, ad, 248–49, 251–53, 255–56, 275–77, 279 ad atig o immu halth, 71–74 hit aout addig, 73–74 ip ad, 100, 161, 163, 280, 282–84, 287, 290 ou o, 73–77, 85 upplmt ad, 84–87, 329–30 axity, 34, 145, 150, 196, 257 glut ad, 61–62 pimopau ad, 60–62
351
t ad, ad, 17, 110, 130, 132, 135, 145, 147–48 toxi ad, ad, 235, 246, 266 appl app l id viga viga,, 90, 185, 207 appl, app l, 39, 76, 79, 203 dtox ad, 254, 271–72 aahidoi aahido i aid, 53–54 am, 48, 129, 297, 306 aomata, aoma ta, 122, 313 ai, 230, 236, 249, 255, 261 atmiii, 204, 213 athiti, xi, 12, 18, 25, 156, 183, 193, 196, 265, 306, 309, 312 augula, 69, 98–99, 160 ahwagadha, 156 apgillu, 208 athma, 9, 13, 265 autim, ix, 242 autoimmu dia: ai o, 11–34 o blum, 3–4, 6, 320 diago o, 1–5, 9, 22–23, 25, 32, 47–48, 51, 177–78, 183, 191–93, 196, 200, 257–58, 261, 297, 302, 306–7, 312, 315–16 lak o uid appoah to, 21 loatio o, 13–14 mot ommo, 24–34 pval o, ix–x, ix–x, 1, 5–6, 9, 14, 21, 177–79, 244 pvtio o, 7–10, 19, 22–24, 116, 193 ao o o ook o, 1–2, 6–7, 10 val o, x–xii, x–xii, 1–5, 7–9, 14, 23–24, 27–28, 40, 61, 125, 154, 181, 190, 212, 254, 320 ig ad ymptom ymptom o, xi, 1, 3–4, 6–7, 21–34, 37, 40, 45, 47–49, 51–56, 120, 122–23, 177–78, 183–84, 191, 194, 241, 256–59, 293, 297, 301–3, 306, 308–13, 315–17 tatiti o, 1, 5, 7, 14, 22, 47, 122, 183, 244, 301, 309, 312 tigg ad au o, ix–xii, 4, 11, 14–22, 25–26, 28, 37, 39, 42, 47–51, 49, 56, 58, 73, 119–20, 123, 129–30, 134, 176–80, 187–90, 229, 233, 239–47, 249–50, 254, 293–306, 309–11, 313–16 wath, wait, ad do othig appoah to, 22–23 autoimmu auto immu gatiti, 183 autoimmu/iammatoy ydom idud y adjuvat (AsIA), 242
352
idx
autoimmu auto immu thyoid dia, 48, 58, 61, 120, 183, 240–41, 293, 296, 314, 319 tatmtt o, 315, 318–19 tatm see also Gav’ dia; di a; Hahimoto’ thyoiditi autoomi auto omi vou ytm, 106, 107, 125, 127, 129 avoado, 69, 271 adal glad ad, 154–55 ad atig o immu halth, 80–82 at i, 53, 55, 154, 300–301 ip ad, 158, 163–64, 170–71 atia, ix, 23, 115, 302, 315 amio aid ad, 43–44 autoimmu auto immu dia tigg ad, 16–19, 294–95 i gut, xi, 9, 17–19, 124, 175–89, 192–93, 195–96, 201–4, 210–12, 214–15, 217, 230, 260, 268–69, 293–94, 299, 306, 308–9, 311, 315 halthy immu ytm ad, 11–12 Ms ad, 298–300 rA ad, 309–10 ip ad, 215, 217–19 t ad, 18, 118 aly,, 79, 207, 254, 270 aly glut ad, ad, 15, 45, 65, 68, 180 ail, 80, 160, 276 a, 4, 20, 319 a, 76, 89, 154–55, 196, 210 dtox ad, ad, 254, 270, 276 ood itiviti ad, 68–69 ip ad, 97, 101, 158, 162, 166–67, 215, 217–19, 284–86, 289 see also lgum , 53, 67, 154, 193, 210, 258 dtox ad, ad, 251, 270–71, 274, 277 ad atig o immu halth, 77, 81–82 iammatio iammatio ad atial imala du to, 16–17 toxi ad, 243, 264 t, 76, 210, 220 i, 184, 204, 213 i, 15, 46, 76, 90, 93, 203 dtox ad, 271–72, 276 Ms ad, 300–301 ta-aot, ta-aot , ta-aotoid, 3, 76, 85, 163–64 dtox ad, 255–56, 277 tai hydohloid (HcL), 184, 208–9, 214
vag, 41, 78–79, 81, 149 see also alohol; o; moothi; oda; ta idoatia, 124, 177, 203–4, 211, 214 il, il aid, il alt: dtox ad, 248, 250–51 gut ad, 181–82, 197–99, 205–8, 213–14 liv ad, 207, 230, 246 l-tt o, o, 198, 205, 208 toxi ad, ad, 240, 246 tatmtt ad, 205–8, 213 tatm iomoitoig, iomoi toig, 232, 235 23 5 iopi, 25, 48, 118, 306–7 ith otol pill, 60, 241, 246, 304 ipholl A (bPA), ipho (bPA), 230, 232, 237, 244, 249 lak uat d oil, 55, 86 ladd, 107 loatig, 52, 130, 154 lia dia ad, 25, 34, 47, 306 dtox ad, 274, 278 ood ad, 45, 63–64, 66, 70 glut ad, ad, 15, 45, 48 gut ad, 180, 185–86, 188, 192–95, 197–99, 204, 213–14, 259 toxi ad, ad, 243, 265 lood, x–xi, 11, 14, 16, 34, 195, 282, 298, 303, 311, 315, 318 dtox ad, ad, 248, 251, 253, 255 d lood ll ad, 24, 89, 182, 255, 258 toxi ad, ad, 19, 233–34, 236–38, 241, 255, 258–59 lood pu, 18, 106, 107, 145 high, 8, 21, 38, 74, 112, 156 mid-ody thiqu ad, 125–26 loodtam, 230, 258 glut ad, 46, 50 gut ad, 124, 180, 189, 197 lood uga, uga, 41, 52, 158 adal glad ad, ad, 18, 154 1 54 DHeA ad, 112–13, 122 ad atig o immu halth, 74–78 t ad, 109–10, 113 lood vl, 1, 23, 28, 38, 59, 86, 125, 155, 177 lui, 69, 90, 203, 254 ip ad, 93, 223–24, 282–83 b lymphoyt atiody ll, 11, 18, 52, 57, 115, 117, 118 gut ad, 176, 179, 189–90 lupu ad, 304–5
idx ody tmpatu, tmpatu, 62, 106, 125, 1 25, 149, 241, 314 see also v ok hoy, hoy, 163, 260, 284 o, 18, 66, 118, 154, 236, 255 DHeA ad, 112–13, 122 atu o, 6, 182, 185 t ad, 110, 113 oag oil, 55, 86 ai, 1, 21, 57, 61–62, 66, 82, 86, 150 dtox ad, 251, 255 gut ad, 177, 259 itio ad, 294–95 Ms ad, xi, 14, 29–30, 257, 294, 297–301 t ad, 106–9, 145 toxi ad, ad, 234, 253, 262 ai og, 14, 34, 48 lia dia ad, 25, 306 dtox ad, 273–74, 276 ood ad, 45, 52, 63, 70–71 gut ad, 195, 259 toxi ad, ad, 231, 235, 266 ad, 89, 154, 202, 270 ad atig o immu halth, 77–79 ood ad, 41, 52, 65, 69 akat, 42, 50, 52, 69, 74, 92, 159, 210, 215–16, 278, 280–81 gut ad, 213–14 t ad, 136, 152 see also rip Idx at: a o, ix, 13, 67–68, 118 toxi ad, ad, 243, 245–46 athig, 5, 45, 83, 106 dp o lly, 126, 144–46, 150–51 gut ad, 192–93 lupu ad, ad, 29, 303 lp ad, 150–51 t ad, ad, 116, 123, 144–46, 150–51 toxi ad, ad, 230, 234, 236 ooli, 76, 155, 161 dtox ad, ad, 251, 254, 260, 276 ooli xtat, 301, 306 omlai, omla i, 207 ukwhat, 45–46, 65, 68, 89, 96, 270 ad atig o immu halth, 77, 79 ug, 166–68, 220–21, 289 utt, 53, 66, 68, 270 see also gh utyat, utyat , 210 21 0 aag, 155, 210, 290 dtox ad, ad, 254, 260, 276
353
admium, 230, 249, 252–53, 255, 261 ai, 79, 149, 158, 193 dtox ad, 270–71, 273 ak, 52, 65, 77–80, 202, 272 alium, 66, 85, 154, 163, 182 aloi, 38–42, 152, 202 a, ix–x, 1, 13, 23, 40, 52, 59, 73, 111, 118, 152, 276 oy ad, 67–68 toxi ad, ad, 229, 236–37, 246 adida, 188, 199, 259, 300 ady, 52, 78, 80, 202, 272 api aid, 202 aohydat, aohyda t, 43, 52 gut ad, 182, 196–97 ip ad, 93, 164 aiog, aiog , 233 aotoid, aot oid, 76, 277 ai, 66, 68, 203, 270 ahw, 224–25 auliow,, 76, 155, 223–24 auliow dtox ad, 260, 276 cD, 326–27 lp ad, 150–51 t ad, 145–47 lia dia, 13, 24–26, 49, 58, 72, 293, 296 ovtioal ov tioal mdii ad, 21–22, 47–48 diago o, 47–48, 51, 306–7 glut ad, ad, 25–26, 47–49, 51, 61, 63, 186, 275, 306–7, 318 gut ad, 25, 47–48, 51, 183, 186, 306–8, 318 ymptom o, 25, 32–34, 47–48, 306 tt o, o, 25–26, 48, 51, 61, 72, 258, 275, 306–8 tatmt o, 307–8 tal vou ytm, 106, 235, 301 Ms ad, 29, 297–98, 300 al, 52, 65, 69, 202, 210, 270 ip ad, 95–96 opial uid (csF), 298 hk, 29, 33, 303 h, 66, 68, 82, 203, 270–71 hlatio,, 240, 262 hlatio i movig havy mtal om ody, 248, 252–56, 260–61 hmial, 9, 38, 62, 65, 73, 87, 121, 234, 256 otio tw autoimmu dia ad, 19, 243–44 liv ad, 20, 230 toxi ad, ad, 19–20, 229–30, 232–33, 237–39, 243–44
354
idx
hik, 43, 69, 90, 154, 210, 258 dtox ad, 270, 276–78 ip ad, 96–97, 101, 158, 165n, 165n, 169–70, 215, 222–23 toxi ad, 243, 264 hikpa, 68, 270 ip ad, 166–68, 285–86 hild, x–xi, 47, 223, 308 gut ad, 178–79, 188 t ad, ad, 110, 116, 127, 131, 133–35 toxi ad, ad, 229, 233–34, 236–38 vai ad, ad, 242–43 Chlamydia pneumoniae, 298–300 hlolla, 252–53 hloi, 234, 237–38, 245 hoolat, 58, 69, 80, 83, 207 dtox limiatio limiatio dit ad, 270, 272 27 2 ip ad, 102, 170–71 holtol, holt ol, 86, 154, 158, 203, 269 DHeA ad, 113, 122 ad, 253–54 hoi autoimmu thyoiditi, see Hahimoto’ thyoiditi ilato,, 253, 276, ilato 27 6, 283 itu ioavooid, 85 oout podut, podut, 90, 154, 215 dtox limiatio dit ad, 270–72 ad atig o immu halth, 74, 79–82 at i, 53, 55 ood itiviti ad, 66, 68–69 gut ad, 202–3, 207, 210 Ms ad, 300–301 ip ad, 169–70, 220 od, 155, 168–69 o, 42, 77–79, 81, 154 dtox limiatio limiatio dit ad, 270–71, 273 27 3 ogitiv utio, 110, 195, 234 sjög’ ydom ad, 31, 312 old, 9, 138 old o, 9, 123 oliti, ix, xi, 2 ollad g, 66, 76, 155, 284 dtox ad, 251, 276 odimt, odimt , 80, 271 27 1 ojugatio atio, 276 otipatio, otipa tio, 34, 110, 225, 241, 253, 265 ood ad, 45, 48, 63, 71 gut ad, 180, 183–86, 188, 195, 197, 259 sjög’ ydom ad, 183–84 ovtioal ov tioal mdii, x, 8, 21–24, 60, 112, 192, 312 appoah to autoimmu dia y, 4–6, 21–23
lia dia ad, 21–22, 47–48 utioal v., 6, 22 mdiatio i, 4–5 ookook, 326 ooki, 39, 41–42, 52, 102, 202, 272 ad atig o immu halth, 77–80 glut ad, 65–66 opp,, 249, 252, 256, 277 opp o, 53, 63, 268–71 dtox limiatio limiatio dit ad, 270–71, 274 27 4 ad atig o immu halth, 72, 77, 79–82 limiatio om dit o, 64–65, 67–68, 184, 189, 193–94, 270–71 gti modiatio (GM) o, 15, 64, 67 gut ad, 184, 189, 193–94 ip ad, 91, 164 itodutio itod utio o, 70–71 otiol, 154, 158, 305, 330 ootig lvl o, 120–21 t o high, 110 t ad, ad, 17, 106–20, 122, 129–30, 137, 140 kill ll, 116–19 omti, 233, 236–37, 272 ough, oughig, 184, 265 amp, 33, 180, 185–86, 195 avig, 17, 94, 105, 110, 138, 152, 218, 221, 266 mii muhoom, muhoom, 155, 165 1 65 umi, 80, 162, 166–67, 276, 283 uumi, 311 yti, 250–51 ytohom P450 ytm, 246, 275 ytotoxi ll, see lymphoyt kill ll daiy podut, 16–17, 53, 154, 268–70 dtox limiatio limiatio dit ad, 270, 272, 272 , 274, 276 ad atig o immu halth, 72, 76, 79, 81–82 limiatio om dit o, xi, 64–66, 68, 184, 189, 193–94, 258, 270, 272, 274, 276 gut ad, 184, 189, 193–94, 203, 207, 210 Ms ad, 300–301 ip ad, 91, 100, 225, 280 itodutio itod utio o, 70–71 itivity to, to, 63–66, 70–71 toxi ad, ad, 243, 264, 272
idx dadlio, dadlio oot, 207–8 dadlio, dath, 1, 28, 184, 231 i gil ad wom, 7 t ad, ad, 105, 113, 131–34 dglyyhiziatd lioi (DGL), 206, 332 dmult,, 207 dmult dditi ll, 176 dtal amalgam (ilv) (ilv) llig, xi, 4, 19, 233–34, 240, 254, 263, 273, 318–19 dpio, dp io, ix, 8, 21, 34, 34 , 48, 74, 151 gut ad, 192, 196 rA ad, 30, 308 t ad, ad, 17, 110, 135, 138, 147 toxi ad, ad, 235, 238, 266 dt, 69, 91–92, 159, 216, 272 ad atig o immu halth, 74, 80 see also rip Idx dtiatio dtiatio pogam pogam, , 327–28 dtox limiatio limiatio dit, 269–76, 278–79 gttig ady o, 273–74 dtoxiatio, xi, 4, 22, 141, 245–56, 304 tog ad, ad, 276–77, 284–85, 290, 305–6, 313 ood ad, 250–54, 260, 269–78 havy mtal moval i, 61, 247–56, 259–61, 277, 283, 286 liv ad, 20, 161, 260–61, 269, 272–80, 290, 293 lupu ad, ad, 305–6, 313 mdial, 277–78 mtaoli, 272, 278–79 atio to, to, 202, 256 ip ad, 161, 163, 280–81, 283–86, 290 l-amt to dtmi d o, 262–68 upplmt uppl mt ad, 250–53, 260–62, 269, 271, 274, 276–78, 330, 332–33 dtox hak, 278–79 DHA, 55, 86, 301, 330 DHeA, 112–13, 330 adal atigu ad, ad, 17, 156–57 lupu ad, ad, 122–23, 156, 15 6, 303–6 sjög’ ydom ad, 312–13 t ad, 113, 122–23 diat, ix, 40, 52, 56, 74, 88, 111, 152, 202, 229 diat, typ 1, 24, 33–34, 183 diaha, xi, 14, 48, 110 lia dia ad, 25, 34, 47, 306 ood ad, 45, 63, 70–71 Gav’ dia ad, 26, 316
355
gut ad, 180, 185–86, 188, 192, 195–96, 198, 213 dihloodiphyltihlo dihloodiph yltihlootha otha (DD), 229, 243–44 dit, dit, ix–xi, 6, 10, 15–18, 20, 43, 47 autoimmu auto immu dia tigg ad, 16–18, 21 dtox ad, 269–76, 278–79 limiatio o o om, 64–65, 67–68, 184, 189, 193–94, 270–71 limiatio o daiy om, xi, 64–66, 68, 184, 189, 193–94, 258, 270, 272, 274, 276 limiatio o glut om, om, xi, 4, 26, 37, 39, 48–51, 61–62, 64–66, 72–73, 184, 189, 193–94, 212, 258–59, 261, 270, 272, 274–75, 307–8, 318 limiatio o oy om, 64–65, 67–68, 184, 189, 193–94, 270, 276 limiatio o uga om, 52, 59, 74–81, 154, 202, 259, 268, 270–71, 273 at i, 52–55, 59 ad ood a iomatio, 37, 42 o wight lo, 40–41 see also ati-ia ati-iammatoy mmatoy dit; limiatio dit digtio, xi, 8, 15–16, 25, 106, 107 dtox ad, ad, 269, 274, 278 gut ad, 180–82, 184–86, 188–90, 193, 195, 197–202, 204–9, 213–14 ip ad, 217, 220 l-amt l-am t o, o, 195, 197–99, 207–8 tatmtt o, tatm o, 200–201, 204–9, 212–14, 331 digtiv ytm, 21, 83, 130, 151, 236, 245, 260 tog ad, 246–49, 254 ood itiviti ad, ad, 63, 67, 70 liv ad, 230–31, 246–49, 252, 256 ip ad, 95, 163 a iv, 181, 199 sjög’ ydom ad, 31, 312 see also gut diidolylmtha (DIM), 277, 306, 332 diidolylmtha 2,3-dimapto-1-popauloi aid (DMPs), 254, 256 dimaptouii dimap touii aid (DMsA), 254–56, 259–60 di, 69, 83, 92, 149, 152, 159, 176, 216, 278, 281 ad atig o immu halth, 74, 80 gut ad, 207, 210, 213–14 see also under et under et i rip Idx
356
idx
dioxi, 230, 237–38, 244, 253, 288 dizzi, 34, 138, 151, 264 DnA, 73, 313 lupu ad, ad, 28–29, 302–303 302 –303 toxi ad, ad, 19–20, 234 dy, 31–33, 110, 183–84, 312 dyioi: diago o, 186, 192, 213 gut ad, 185–88, 190, 192–96, 200–206, 211–14, 294, 311 laky gut ydom ad, 186–88, 200, 212–13 rA ad, 309, 311 l-amt o, 195–96, 200–202 ymptom ympto m o, 185–86, 213 tatmtt o, 192–94, 200–206, 211–14, tatm 331 typ o, 185–86 a, a itio, 12, 66, 265 eA tt, 305 atig: o immu halth, 71–82, 75 midul, 41, 82–84, 152, 268 see also dit, dit; ood, ood ebnA tt, 305 damam, 68, 270 eDA, 254–56 ggplat, 64, 76, 193, 311 gg, 64, 69, 155, 210 dtox ad, ad, 251, 270, 274 limiatio om om dit o, 189, 193, 270, 274 iammatio iammatio ad atial imala du to, 16–17 ip ad, 160–61 toxi ad, 238, 243 lutho (siia gig), 155 limiatio dit, 81, 88, 311 ood itiviti ad, 63–72 gut ad, 184, 189–90, 193–94, 200, 203, 207 mu ida duig, 69 see also dtox limiatio dit doi glad, doi ytm, ix, 60, 108, 109, 237 gy, 4, 17, 41, 78, 83, 120, 249–51 dtox ad, 251, 261 gut ad, 193, 195–96 ip ad, 158, 166 t ad, ad, 121, 137–38, 153 t, see rip Idx
viomt, 47, 63, 125, 295–96 viomt, gut ad, 178, 186 lupu ad, ad, 301, 305 rA ad, 309–10 t ad, ad, 135, 139, 144–47 toxi ad, ad, ix, 6, 19–20, 121, 229–30, 232–33, 236–40, 243–44, 246–47, 249–50, 252, 256, 262–63, 272–73, 279, 293, 295–96 zym, 56, 58, 157, 218, 331 dtox ad, ad, 20, 248–50, 256, 278–79 ood ad, 41–42 gut ad, 180–82, 184–85, 188, 197, 199, 205–8, 213–14, 231, 268 liv ad, 20, 231, 248–49, 256, 279, 305 toxi ad, ad, 20, 231, 234, 247 ePA, eP A, 55, 86, 330 pigallaathi pigallaat hi gallat (eGcG), 59, 77, 87, 277, 329 epti-ba ept i-ba viu (ebV), 8–9, 21, 118, 123, 315 lupu ad, ad, 302–5, 310, 310 , 313 Ms ad, 299 rA ad, 310–11, 313 sjög’ ydom ad, 313–14 ythoyt dimtatio dimtatio at (esr), 30–31, 89, 308–9 tial atty aid, 54–56, 67, 81, 311 xampl xamp l o, 54–55, 86 86 ip ad, 93–94, 99, 288 tog, 118, 157 dtox ad, ad, 276–77, 284–85, 290, 305–6, 313 DHeA ad, 113, 122 digtiv ytm ad, 246–49, 254 liv ad, 20, 22, 246–47, 249, 305 lupu ad, ad, 28, 246, 302–6 pimopau ad, 60, 62 ip ad, 284–85, 290 sjög’ ydom ad, 312–13 t ad, 114, 122 toxi ad, ad, 20, 230, 237, 243–46, 290, 306 xotog ad, 230, 243–47, xotog 305 vig pimo oil, 55, 86 xi, x, 3, 6, 149, 258–60 dit ad, 40, 273 t ad, ad, 17, 105, 113, 121, 132, 136–37, 142, 152–53 toxi ad, ad, 235, 262
idx y, 64, 84, 107, 110, 146 Gav’ Gav ’ dia ad, 26, 241, 315 Ms ad, 29, 33, 297 sjög’ ydom ad, 31, 33, 183–84, 312 toxi ad, ad, 235, 265 amili, amily, amily, 6, 9, 37, 37 , 63, 129, 296, 296 , 320 t ad, ad, 105, 130–31, 133–35, 149 at, ody, xi, 38, 90, 198, 251, 269 lly,, 78, 110–11 lly toxi ad, ad, 238, 243, 262 at, ditay, ditay, 47, 258, 302 adal glad ad, 154–55 ad, 52–54, 59, 81–83, 121, 220, 268, 270 dtox limiatio dit ad, 270–71 ad atig o immu halth, 72, 81–82 ood ad, 17, 39, 42–43, 52–55, 66–69, 86, 99 good, 53–54, 59, 72, 81–82, 84, 86, 154–55, 158, 160, 168, 170–71, 247, 268, 300–301 gut ad, 179–80, 182, 194, 197–98, 207–8, 210 iammatio iammatio ad, 39, 42, 53–56, 67, 93, 99, 194 Ms ad, 300–301 patially hydogatd, hydogatd, 53, 81–82, 271 ip ad, 99–100, 102, 158, 160, 168, 170–71, 220, 223, 288 atuatd, atua td, 52–55, 67, 81–82, 100, 219, 268 ou o, 42, 53, 55, 81, 86, 154, 300–301 t ad, 110, 121 upplmt uppl mt ad, 53–56, 84–86, 271 ta, 42, 52–54, 81–82, 268, 271 vgtal ad, ad, 55, 81–82, 86 see also tial atty aid atigu, x–xi, x–xi, 1, 3, 8, 14, 21, 32, 78, 129, 241, 258–59, 302, 314 lia dia ad, 25, 47, 306 dtox ad, 273–74, 276–77 ood ad, 45, 70 glut ad, 45, 48 gut ad, 186, 191–92, 196, 202, 204, 213, 259, 294 Hahimoto’ thyoiditi ad, 27, 317 rA ad, 30, 308 sjög’ ydom ad, 31, 312
357
t ad, ad, 17–18, 105, 118, 120–21, 131–34, 137–42, 153, 156–57 toxi ad, ad, 231, 235, 238, 265 t, 33, 110, 191 gut ad, 188–89 lupu ad, ad, 29, 303 Ms ad, 257–58 toxi ad, ad, 231, 235 l, 220 tu, 234, 237–38, 253 v, 12, 21, 294 lupu ad, ad, 28–29, 32, 302 rA ad, 30, 308 , 15, 17, 77, 154, 252, 305 i dtox, 253–54 gut ad, 186, 196, 202–3, 205, 211, 253–54 ip ad, 102, 224, 280, 285–86 a, 127, 133–35 g, 231 gut ad, 191, 193 lupu ad, ad, 29, 33, 303 rA ad, 30, 191, 308 h, 69 adal glad ad, 154–55 dtox ad, ad, 253, 271, 277–78 ad atig o immu halth, 81–82 at ad, 53, 55, 86, 99 ip ad, 99–100, 221, 288 toxi ad, ad, 4, 20, 82n, 82n, 121, 233–34, 238, 244, 254, 258, 263–64, 275, 319 h oil, 53–56, 82, 86, 271, 305, 330 axd, axd oil, 53, 56, 82, 86, 157, 207, 254, 271, 305, 313 ou, 67, 77, 89 ip ad, 91, 93, 96, 100 see also whit ou u, 9, 12, 138, 314 olat,, 62, 332 olat dtox ad, 276–77 ip ad, 96, 287 ood allgi, 38–39, 44–45, 68 lia dia ad, 25, 306–7 ompaio ompaio tw ood itiviti ad, 62 glut ad, 15, 25, 49, 180 gut ad, 124, 186, 188, 192, 199, 210 t ad, 124–25 ood, ood, x–xi, 2, 7–9, 11, 115, 130, 146, 175, 178–82, 214, 293, 296, 304–7, 327 o adal glad, 17, 143, 154–55 autoimmu auto immu dia tigg tig g ad, 18–19
358
idx
ood, ood (cont. (cont.)) atial imala du to, 16–18 avig ad, ad, 17, 94, 105, 110, 138, 152, 218, 221, 266 ultud o mtd, 165, 203, 206–7, 210 dtox ad, ad, 250–54, 260, 269–78 at, 46, 52, 264 at ad, 17, 39, 42–43, 52–55, 66–69, 86, 99 utio o, 38–39 gti ad, 15, 37, 41–42, 46–47, 54, 63–64, 67–68, 154, 305 glut ad, ad, 15, 25–26, 34, 39, 45–52, 49, 62–66, 68, 70–71, 180, 306–7 gut ad, 18–19, 44–45, 66, 124, 180–82, 184–90, 192–95, 197–203, 206–8, 210–13, 259, 268–69 immu utio ad ala impovd y, 53–59 iammatio iammatio ad, 16–18, 37–42, 44–45, 52–56, 59, 62, 67, 71–72, 151, 194, 203, 268–69, 301 a iomatio, iomatio, 37, 40–43 lal ad, 65, 67, 77, 79, 81, 270–71, 278 liv ad, 20, 230 a mdii, 5, 7, 37–102, 154–55, 201, 253, 269–77, 300 Ms ad, 300–301 pod, 15–17, 46–47, 52–53, 67, 269, 280 rA ad, 37, 45, 53–56, 311 ltio o, 39–40, 59, 82–83 lp ad, 149, 151 t ad, ad, 105, 110, 113–14, 121, 124–25, 132, 136–38, 141–43, 152, 158, 268–69 txtu ad avo avo o, 83–84, 94, 97, 101–2, 168, 170–71 that d to movd, 43, 45, 48–53 toxi ad, ad, 19, 230, 236, 238, 243, 247, 262, 264–66, 276–77 whol, xi, 16, 66, 280 see also dit, dit; rip Idx ood itiviti, 37, 39–40, 44–45, 59, 62–73, 311 dtox ad, 269–70, 275 ad atig o immu halth, 72–73, 81 glut ad, ad, 25–26, 34, 39, 48–50, 49, 68, 70–71, 306 gut ad, 124, 182, 186, 188, 193, 200
iammatio ad, 45, 62, 67, 71–72 iammatio t ad, 124–25 ttig o, 63–72, 81 adial, 58, 234, 275 atioxidat ad, 73, 85 glutathio ad, 249–51 3, 4: Gav’ dia ad, 26–27, 316 Hahimoto’ thyoiditi ad, 27–28, 61, 317 ittata, 160–61 utooligoahaid (FOs), 203, 205, 211 uit, 3, 39, 43, 52, 54, 69 dtox ad, ad, 254, 270–72, 274, 276, 278 ad atig o immu halth, 72–76, 79–80 gut ad, 203, 207 ip ad, 93, 95–96, 158, 163–64, 169–71, 215, 218, 220–21, 224–25, 280, 282–83 upplmt ad, 85, 87 utioal mdii, x–xii, 1–2, 4–6, 8, 10, 14, 22–24, 60, 71–72, 121, 125, 128, 293, 305, 311 dtox ad, 254, 279 gut ad, 176–77, 191, 201, 205, 209, 212 Ms ad, 257, 300–301 poioal po ioal hlp, hlp, 88–90, 151–52, 156 gallladd, 181–82, 198 gallladd, gamma lioli aid (GLA), 55, 86, 311, 330 gali, 80, 155, 207 dtox ad, 251, 276 gut ad, 203–4 ip ad, 165, 220, 222, 282–85 ga, 15, 52, 130, 154, 278 lia dia ad, 25, 34, 47, 306 ood ad, 45, 63–64, 66 gut ad, 180, 185–86, 188, 192–95, 197, 199, 202, 204, 213–14, 259 gatoophagal gatoop hagal ux dia di a (GerD), 199, 207 g, gti, ix, 4, 57, 188, 295–96, 298, 304, 308 lia dia ad, 47, 307 dtox ad, 250, 252 ood ad, 15, 37, 41–42, 46–47, 54, 63–64, 67–68, 154, 305 glut ad, 46–47, 50 lupu ad, 301, 309
idx rA ad, 309–10 toxi ad, ad, 19, 231, 234, 247, 262, 267–68 gh (laid utt), 55, 82, 90, 210, 271 ip ad, 164–65, 215, 219, 223, 225 gig,, 80, 163–164, 209 gig gliadi, 25, 46, 51, 72, 275, 307 gluo, 41, 78, 197 glutami aid, 250 glutami, 332 gut ad, 193, 210–11, 214 ip ad, 215, 221–22 glutathio, 58, 282, 293, 301, 319 i movig havy mtal om ody, 248–53 glut, 22, 89, 154, 268–70 altativ alta tiv to, 65–66, 68 autoimmu auto immu dia tigg ad, 14–16, 25–26, 39, 47–51, 49, 293–95 lia dia ad, 25–26, 47–48, 49, 51, 61, 63, 186, 275, 306–7, 318 dtox limiatio dit ad, 270, 272, 274 ad atig o immu halth, 72–73, 77, 79 limiatio om om dit o, xi, 4, 26, 37, 39, 48–51, 61–62, 64–66, 72–73, 184, 189, 193–94, 212, 258–59, 261, 270, 272, 274–75, 307–8, 318 ood ad, 15, 25–26, 34, 39, 45–52, 49, 62–66, 68, 70–71, 180, 306–7 gut ad, 25, 46, 48, 50, 180–81, 184, 186, 189–90, 193–94, 202, 210–12, 294, 306, 308 Hahimoto’ thyoiditi ad, 4, 318 ip ad, 91, 94–96, 102, 169, 283 itodutio itod utio o, 70–71 ymptom o itivity to, 48–50 glymi idx, 52, 72, 74–77, 79–80, 88 glyi, 250, 278 goit, 27, 317 gai, 89, 202–3 dtox ad, ad, 254, 270, 272 ad atig o immu halth, 74–80 ood itiviti ad, 65, 67–68 gti modiatio (GM) o, 15, 64, 67 glut ad, 15, 45–47, 49, 65, 180 Ms ad, 300–301 ip ad, 91, 93, 95–98, 283 whol, 77, 79, 89, 96, 154, 202, 254, 270, 272 gaola, gaola a, a, 69, 91, 94–95, 202, 217–18
359
gapuit d xtat, 204 gap, 42, 76, 83, 276 Gav’ Gav ’ dia, 2, 7, 14, 20–22, 24–27, 2 4–27, 183, 293–94, 296, 307 diago o, 51, 315 ymptom o, 26, 32–34, 120, 241, 315–16 tt o, o, 26–27, 61, 314, 316 toxi ad, 20, 241 tatmtt o, 315, 318–19 tatm tigg ad au o, 314–15 g dik, 276–77 g ta, 54, 59, 77, 87, 150, 301 guidd imagy, 125–28, 150 t ad, ad, 128, 144–45, 147–48 gut, ix, 22, 38, 52, 141, 258–61, 296 autoimmu auto immu dia tigg ad, 14, 18–19, 21, 25 atia i, xi, 9, 17–19, 124, 175–89, 192–93, 195–96, 201–4, 210–12, 214–15, 217, 230, 260, 268–69, 293–94, 299, 306, 308–9, 311, 315 lia dia ad, 25, 47–48, 51, 183, 186, 306–8, 318 dtox ad, ad, 255–56, 260, 275 dyioi ad, 185–88, 190, 192–96, 200–206, 211–14, 294, 311 limiatio dit ad, 184, 189–90, 193–94, 200, 203, 207 ad, 186, 196, 202–3, 205, 211, 253–54 ood ad, 18–19, 44–45, 66, 124, 180–82, 184–90, 192–95, 197–203, 206–8, 210–13, 259, 268–69 utioal mdii ad, 176–77, 191, 201, 205, 209, 212 glut ad, ad, 25, 46, 48, 50, 180–81, 184, 186, 189–90, 193–94, 202, 210–12, 294, 306, 308 halig o, 7, 19, 72, 164, 175–225, 258, 261, 275, 293, 304, 306, 308 itio ad, ad, 18–19, 181–83, 186, 188, 190, 199, 294–95, 309 lik tw immu ytm ad, 175–80, 182–88, 194 lupu ad, ad, 183, 306 Ms ad, 185, 259, 301 rA ad, 183, 185, 309–11 ip ad, 215–25 l-amt o, 195–202, 205, 207–8, 210, 212 t ad, ad, 105–6, 110, 124–25, 179, 181–83, 185–86, 188, 196, 200
360
idx
gut (cont. (cont.)) upplmt uppl mt ad, 177, 184–85, 187, 199, 201, 203–9, 211, 213–14, 259, 331–32 toxi ad, ad, 180, 185–86, 188, 202–3, 230, 245, 259, 268–69, 306 tatmtt o, 192–94, 200–214, 331 tatm see also laky gut, laky gut ydom gut-aoiatd lymphoid tiu (GAL), 18, 177 hai, 192, 241, 314 hai, lia dia ad, 32, 47 Hahimoto’ thyoiditi ad, 27, 32, 317 lupu ad, ad, 29, 32, 303 toxi ad, ad, 234, 240, 265 had, 3, 30, 33, 67, 71, 106, 110, 177, 188–89, 235, 308 Hahimoto’ thyoiditi, 2–4, 7, 13–14, 20–22, 24–25, 58, 129, 293–94, 296, 307, 314–18 o blum, 3–4, 6 diago o, 3–4, 51, 316 hypothyoidim hypothy oidim ad, 316–18 with omal thyoid utio, 317 ymptom ympto m o, 27, 32–34, 317 tt o, o, 27–28, 61, 194, 314, 317–18 toxi ad, 4, 20, 241 tatmtt o, 3–4, 27–28, 316, 318 tatm tigg ad au o, 314–15 hadah, 34, 38, 126, 193 dtox ad, ad, 256, 273–74, 276 ood ad, 52, 63, 70 glut ad, 45, 48 gut ad, 189, 202, 204 t ad, 105, 110 toxi ad, 231, 264 hat, 28, 41, 88, 107, 125, 153, 155, 192, 242, 265, 314 Gav’ Gav ’ dia ad, 26, 120, 241, 241 , 315–16 t ad, ad, 105–9, 145, 147 hatu, 15, 25 gut ad, 180–81, 183–85, 196–97, 204, 207–8 hat dia, ix, 1, 38, 40, 52–53, 59, 74, 99, 111, 202, 229 havy mtal, xi, 9, 73 autoimmu auto immu dia tigg ad, 14, 19, 233, 239–43, 247, 249–50, 254 ip ad, 283, 286 moval om ody o, 4, 61, 247–56, 259–61, 277, 283, 286
toxiity ad, ad, 230, 233–37, 239–44, 2 39–44, 249, 254–55, 258–59, 262, 275, 279 Helicobacter pylori, 183, 315 hmp, 66, 68, 90, 270, 276 hpatiti, 9, 123, 242, 315 h, xi, 80, 121, 311 adal glad ad, ad, 155–56, 330 dtox ad, ad, 260–61, 271, 275–76, 279 guid to, 329–33 gut ad, 178, 184, 192–93, 201, 203–6, 211, 213–14 ip ad, 99–100 lp ad, 150–51 t ad, 141, 330 high-itivity c-ativ pot poti i (cadio crP), 30–31, 88, 308–9 hiv, 45, 186, 241, 265 HLA ttig, 296, 307 hoi, 136–37, 145 homo plamt, 304 tog ad, ad, 244, 246 thyoid ad, 3–4, 27–28, 316, 318 homo, 26–28, 58, 87, 151, 194, 300 autoimmu auto immu dia tigg ad, 14, 17–18 Gav’ dia ad, 61, 120, 241, 314–15 Hahimoto’ thyoiditi ad, 3–4, 27–28, 241, 316–18 lupu ad, ad, 304–6, 313 pimopau ad, 60–62 ip ad, 99, 169–71, 280 t ad, ad, 14, 17–18, 22, 106–23, 125, 129–30, 137, 140 toxi ad, ad, 20, 237–38, 243–44, 275 vitami D ad, ad, 56–57 see also tog; ttoto hot ah, 70, 110, 149, 151, 265, 274 glut ad, 50, 61–62 pimopau ad, 60–61 hummu, 69, 79, 272 hug,, 46, 64, 81, 83, 110, 273, 282 hug hut-gath, 15, 46 5-hydoxytyptopha 5-hydo xytyptopha (5-HP), 151 hygi hypothi, 178 hypthyoidim, see Gav’ dia hypohlohydia, hypohlohy dia, 183, 199, 208–9 hypothalamu, 108, 109–10 hypothyoid, hypothy oid, hypothyoidim, ix, 3–4, 28, 314, 316–18 i am, 69, 80, 202, 272 immu omplx, immu-omplx dia, 16, 45, 55, 189, 309–11
idx immu ytm: ala o, x–xi, x–xi, 1, 9–12, 19, 21, 52–59, 84, 87–88, 112, 130, 134, 155, 176, 191, 293, 312 go awy, 13 hadiappd, hadia ppd, 295–96 halig o, 10, 14, 17, 19, 52, 88, 154 halthy,, 11–13, 17, 19, 21–22, 71–82, halthy 75, 84, 152, 167, 190 immuomodulato, immuomodula to, 87, 114–15, 118 immuouppiv dyioi, 185–86 idol-3-aiol, idol-3-a iol, 277, 284, 290, 306 iat, 178–79, 242 toxi ad, ad, 233, 238 itio, itio, x, 3, 32, 44, 175, 178–79, 313 o a, 12, 66 ood ad, 37, 66 gut ad, 18–19, 181–83, 186, 188, 190, 199, 294–95, 309 halthy immu ytm ad, 11–12, 22 lik tw autoimmu dia ad, 14, 18–21, 293–97 lupu ad, 302–5 Ms ad, 294, 297–99 rA ad, 309–11 t ad, ad, 115, 118–19, 121, 123–24 upplmt ad, 85, 88 vai ad, ad, 242–43 itiou moouloi moouloi (moo), 8, 21, 21 , 118, 299, 303–4, 314 iammatio, iammatio , iammatio iammatio, , ix–xi, 11–14, 23, 47, 52–59, 86, 116, 117, 122, 175, 235, 242, 295, 315 autoimmu auto immu dia tigg ad, 16–18, 21, 42 dit ad, 40, 53–54, 59, 72–82, 88, 268–69, 275, 302 at ad, 39, 42, 53–56, 67, 93, 99, 194 ood ad, 37–42, 44–45, 52–56, 59, 62, 67, 71–72, 151, 194, 203, 268–69, 301 glut ad, 16, 50 gut ad, 38, 124, 177, 184, 189–90, 194, 199, 203, 207, 210, 259 Hahimoto’ thyoiditi ad, 4, 27, 317 halthy immu ytm ad, 11–12 lupu ad, 301–2 Ms ad, xi, 297, 301 rA ad, 30–31, 191, 308–9, 311 ip ad, 93, 99, 101, 163, 223, 283 sjög’ ydom ad, 183–84 t ad, ad, 109, 111, 114–15, 119–20, 124, 138
361
ad uppot om utioal mdii halth poioal, 88–89 iammatoy iammat oy dyioi, 186 iat immu ytm, 175 iuit good atia, 185 iuli, 56, 78, 88, 110 ittial pmaility tt, 212 itti, 107, 118, 180 see also gut iuli, 203, 205, 211 iital owl ydom, ydom, 110, 124, 185 ithig, 30, 33, 265, 297 gut ad, 196, 198 joit, ix–xi, 12, 295 lia dia ad, 33, 48 ood ad, 45, 52, 63, 70, 214, 274 glut ad, ad, 45, 48, 50 gut ad, 177, 186, 189, 191, 194, 214 lupu ad, 28–29, 301, 303, 309–10 pai i, 1, 14, 16, 23, 28–29, 31, 33, 37–38, 45, 48, 52, 63, 70, 114, 120, 134, 138, 154, 183–84, 186, 189, 193, 214, 265, 274, 303, 308, 311–12 rA ad, 23, 30, 33, 37, 120, 191, 308–11, 314 sjög’ ydom ad, 31, 33, 183–84, 309, 312, 314 t ad, ad, 114, 120, 134, 138 toxi ad, ad, 262, 265 joualig, 83–84, 128, 144, 146, 151 jui, 207, 271, 276, 286 ad atig o immu halth, 78–79 kal, 55, 66, 76, 155, 260, 276 ip ad, 160–61, 163–64, 284, 286 kamut, 79, 270 glut ad, ad, 15, 45, 65, 68, 180 k,, 66, 68, 217, k 2 17, 270 gut ad, 202–3, 207 kthup, 80, 271 kto, 300–301 kidy, 17, 21, 24, 112, 310 dtox ad, ad, 248, 251–52, 259 lupu ad, ad, 23, 28, 301 toxi ad, ad, 230, 234, 258 kithai, 219–20 laimal glad, 31, 312–13 latoailli, 124, 177, 203–4, 211, 214, 308 laui aid, 202
362
idx
lad: moval om ody o, 252–55 toxiity ad, ad, 230, 236–37, 240, 263, 279 laky gut, laky gut ydom, 182, 186–90, 197, 293, 308, 314 au o, 124, 187–88, 200 diago o, 192, 213 dyioi ad, 186–88, 200, 212–13 ood ad, 44–45, 62 ittial atia ad, 180–81 lik tw immu ytm ad, 187–88 rA ad, 309, 311 l-amtt o, 195, 199–200, 210 l-am tgthig ittial liig o, 210–13 ymptom ympto m o, 188–90, 213 tatmtt o, 194, 200, 210–13 tatm lg, 6, 48, 106, 119, 193, 297, 306 lgum, 89, 154, 217 dtox ad, ad, 254, 270, 277 gut ad, 203, 210 see also a lmo, 69, 163–64, 285–86 ltil, 89 dtox ad, ad, 254, 270, 276 ood itiviti ad, 68–69 ip ad, 218–19, 283–84 L-glutami, L-glutami , 211, 214 lioi, 156, 206, 332 lim, 162, 169–70 lipa, lipa, 197, 207–8 liptik, 236–37 liv,, x–xi, 4, 17, 21, liv 21 , 41, 56, 207 dtox ad, ad, 20, 161, 260–61, 269, 272–80, 290, 293, 332–33 digtiv ytm ad, 230–31, 246–49, 252, 256 zym ad, 20, 231, 248–49, 256, 279, 305 tog ad, 20, 22, 246–47, 249, 305 havy mtal moval ad, 249, 252, 255–56, 260 ip ad, 280–90 uppot o, o, 7, 247–49, 256, 258–60, 268–90, 293 toxi ad, ad, 20, 229–31, 238–39, 246–47, 258, 262, 267, 269, 280 tatmt pogam o, 268–79 logvity g, 42, 54 low-uga dit, 72, 74–81, 88 low-uga LpPLA2, 88
luh, 69, 92, 145, 152, 155, 159, 216, 278, 281 see also under et under et i rip Idx lug, 21, 31, 107, 118, 146, 178, 234–35, 255, 312 lupu, ix, 2, 7, 13, 19–24, 242, 293, 296, 301–6 DHeA ad, 122–23, 156, 303–6 ebV ad, ad, 302–5, 310, 313 tog ad, ad, 28, 246, 302–6 h oil ad, 56, 305 gut ad, 183, 306 t ad, ad, 119–20, 122 ymptom ympto m o, 28–29, 32–34, 56, 122–23, 301–3, 309–10, 312 tt o, o, 29, 193, 240, 240 , 302–3, 305–6, 309 toxi ad, ad, 19–20, 240, 240 , 244–46 tatmtt o, 303–6, tatm 303 –6, 313 tigg ad au o, 301–5, 310, 313 lymph od, 18, 33, 118 lymphoyt, 11, 115, 117, 176, 312 toxi ad, ad, 240, 244–45 244 –45 see also b lymphoyt atiody ll; specifc cells lymphoid lympho id oga ad tiu, 106, 118 gut ad, 18, 177, 189 magium, 182, 287 maga, maga , 256, 277 mapl yup, yup, 79, 164, 271, 288 mat, 43, 67, 268, 289 dtox ad, ad, 251, 270, 278, 2 78, 300 ad atig o immu halth, 76, 81–82 toxi ad, ad, 238, 243, 243 , 272 mdiatio, mdiatio , ix–xi, 9–10, 21–24, 37, 60, 122, 194, 243, 315, 320 otiol ad, 120–21 dtox ad, 252, 275 gut ad, 18, 124, 178–79, 182–84, 186, 188, 192, 196, 204, 207, 211, 299 Hahimoto’ thyoiditi ad, 27–28, 316, 318 Ms ad, 298–300 rA ad, 191–92 id t o, ix–x, 5, 9, 21–22, 129, 151, 184, 192, 299 lp ad, 21, 150–51 t ad, 114, 116, 120, 124 o ymptom otol, xi, 4, 21–22, 56 toxi ad, ad, 230, 238–39, 238 –39, 264
idx mditatio, 3, 83–84, 125, 127–28, 150 t ad, ad, 128, 132, 136, 144–45, 147–48 mmoy,, 110, 171, 235, 238, 266 mmoy m, 108 ttoto ad, 122, 157 toxi ad, 243–44 mopau, mopau , 60–61, 113, 236, 304 lp ad, 149, 151 t ad, 18, 110 ymptom o, 50, 110, 149, 151 mtuatio,, 60–61 mtuatio t ad, 18, 110 toxi ad, ad, 243, 245–46 mu, 69, 80, 91–92, 159, 215–16, 280–81 muy, x–xi, 73, 121, 273, 302 autoimmu auto immu thyoid dia ad, 240, 318–19 otio tw autoim autoimmu mu dia ad, 239–43, 247, 249–50, 254, 293 dtal amalgam llig ad, ad, xi, 4, 19, 233–34, 240, 254, 263, 318–19 i h, 4, 20, 82n, 82n, 121, 233–34, 254, 258, 263, 319 Hahimoto’ thyoiditi ad, 4, 20, 241 poioig ad, 19–20, 235 moval om ody o, 4, 61, 247–53, 255–56, 259–61 ig ad ymptom o xpou to, 234–35 toxiity ad, 4, 8, 19, 61, 230, 233–36, 239–44, 249, 254–55, 258–60, 263, 279, 293 vai ad, ad, 233, 241–43 muy vapo, 234–35 Mtallocla,, 261, 332 Mtallocla mtallothioi (M), (M), 248–49, 251–52, 261 mthyl doo, 62 mthylmuy,, 234–35, 253, mthylmuy 25 3, 255 milk, 90, 165n 165n dtox limiatio dit ad, 270–71 ad atig o immu halth, 80–82 ood itiviti ad, 66, 68–69 gut ad, 202–3, 210, 215, 224 toxi ad, ad, 238, 243 milk thitl, 277, 329 millt, 65, 68, 77, 79, 89, 270 mid-ody,, 2, 5–6, 151 mid-ody t ad, ad, 125–28, 132, 145, 148 midul atig, 41, 72, 82–84, 152, 268
363
mial, 43, 47, 252 dtox ad, ad, 256, 260, 276–77 ad atig o immu halth, 73–77 gut ad, 182, 199 ip ad, 100–101, 160, 223–24, 280, 285–87 upplmt upp lmt ad, 58, 84–87, 277, 307, 329 ta, 86, 230 mioyli, 299–300 mitohodia, 251, 300 modid itu pti, 205, 211, 332 mold, 11, 19, 43, 175, 178, 208, 218, 263, 275 molula mimiy, 190, 294, 302–3, 310 glut ad, 16, 50–51 mood, 34, 50, 78, 110 gut ad, 192, 202 toxi ad, ad, 246, 266 mouth, 84, 110, 146, 175–76, 318 sjög’ ydom ad, 31, 33, 183–84, 312 ul i, 29, 33 mu, 65, 77, 79, 91, 93, 272 multipl loi loi (Ms), ix, 2, 13–14, 21, 24–25, 56, 58, 130, 242, 293, 296–301, 307 lia dia ad, 48, 51 diago o, 51, 257–58, 261, 297 gut ad, 185, 259, 301 itio ad, 294, 297–99 ymptom ympto m o, xi, 29–30, 33–34, 256–59, 297 tt o, 30, 297, 300 toxi ad, 20, 240 tigg ad au o, 297–301 multivitami/multimial upplmt, 58, 85, 87, 277, 307 mul, ix, 12, 106, 122, 156, 242 dtox ad, 255, 276 ood ad, 45, 52, 63, 214 glut ad, 45, 48 gut ad, 186, 188–89, 214, 294 lupu ad, ad, 28–29, 302–3 pai i, 1, 8, 14, 21, 28–31, 33, 37, 45, 48, 52, 63, 114, 120, 134, 138, 154, 186, 214, 231, 265, 276, 294, 302–3, 308, 312 rA ad, 30, 37, 308 sjög’ ydom ad, 31, 183, 312 t ad, ad, 110, 113–14, 120, 134, 138 toxi ad, ad, 231, 235, 262, 265
364 muhoom, 155 muhoom, xtat ad, 54, 88 ip ad, 101–2, 165–66 myli, 29, 50, 257, 294, 297, 299, 301 n-atylyti (nAc), (nAc), 255–56, 277, 301, 319, 330, 332 aua, 21, 34, 154, 265 gut ad, 185, 198, 213 k, 33, 110, 129, 314 Ms ad, 30, 297 vou ytm, ix, 48, 50, 107, 155, 234 mid-ody thiqu ad, 125, 127 Ms ad, 29, 297–98, 300–301 t ad, ad, 106–9, 114, 116, 118–19, 125, 129–30, 145, 148, 158 iai, 96, 288 opiphi, opi phi, 114, 116, 120, 124 o, 12, 146, 175 lupu ad, ad, 29, 33, 303 um, 48 lia dia ad, 25, 34, 306 dtox ad, 259–61 Ms ad, 29, 34, 257–58, 297 toxi ad, 235, 245 ut utt, 69, 79, 90, 94–95, 102, 170, 272 utigomi, utigo mi, 37, 42 ut, 15, 46, 154 dtox ad, ad, 254, 270–72, 274, 276–77 ad atig o immu halth, 76–77, 79, 81–82 at i, 42, 53, 55, 86 ood itiviti ad, 66, 68–69 Ms ad, 300–301 ip ad, 95–96, 221–24 see also d oatmal, 69, 95, 102 oat, 45–46, 65, 68, 89, 102, 207 dtox ad, 251, 254 oity,, 110–11, oity 110–11 , 153 oliv oil, 53, 258, 270–71 ad atig o immu halth, 74, 82 ood itiviti ad, 68–69 omga-3 at, 54–55, 67, 81, 86, 233 ip ad, 99, 158, 160, 168, 288 omga-3 idx, 89 omga-6 at, 55, 81, 86, 311 oio, 76, 203, 207 dtox ad, ad, 251, 254, 276 ip ad, 220–21, 282–83 oag, 76, 220, 254, 270–71, 274, 278
idx ogao, 80, 184, 204, 213, 259, 331 ogao, ogai podut, podut, 90, 154, 202, 305 dtox ad, 271, 276 ad atig o immu halth, 73–74, 80–81 Ms ad, 300–301 ip ad, 169, 280, 288 toxi ad, ad, 243, 272 ogaohloid, 230, 238, 245–46 ogaophophat, 230, 238 otopooi, 122, 153, 182, 184, 192 ovai, 108, 109, 113, 237 pai, pai, xi, 6, 12, 314, 320 i adom, 14, 34, 48, 183–84, 213 lia dia ad, 33–34 ovtioal ov tioal mdii ad, 5, 23 dtox ad, 273–74, 276 ood ad, 45, 52, 63, 70, 214 glut ad, ad, 37, 39, 45, 48 gut ad, 183–84, 186, 189, 191–93, 213–14, 294 i joit, 1, 14, 16, 23, 28–29, 31, 33, 37–38, 45, 48, 52, 63, 70, 114, 120, 134, 138, 154, 183–84, 186, 189, 193, 214, 265, 274, 303, 308, 311–12 lupu ad, 28–29, 33–34, 123, 302–3 Ms ad, 29, 297 i mul, 1, 8, 14, 21, 28–31, 33, 37, 45, 48, 52, 63, 114, 120, 134, 138, 154, 186, 214, 231, 265, 276, 294, 302–3, 308, 312 rA ad, 30, 33, 37, 120, 308, 311 sjög’ ydom ad, 31, 33, 183–84, 312 t ad, ad, 110, 114, 119–20, 134, 138 toxi ad, ad, 231, 265 pait, 229, 236–37, 263 palo dit, 46, 300–301 paati paa ti zym, 181–82 gut ad, 205, 214 l-tt o, 197, 205 paati, paa ti, 207–8 paty hoppig lit, 89–90 papaya, papa ya, 76, 207–8 paait, 11, 43, 61–62, 115, 175, 230 gut ad, 178, 185–88, 195–96, 199, 203, 205, 209, 212, 260, 294 paaympathti paaympa thti vou vou ytm, 106–9, 107, 119, 145 paait, 224–25 paly,, 80, 99, 160, 207, 210, 222 paly paioow, 150–51
idx pata, 64–65, 69, 89, 210, 270 ad atig o immu halth, 74–79 ip ad, 96–99 pah, 76, 79, 93, 271–72 paut, paut utt, 64, 69, 90, 193 dtox ad, 270–72, 274 pa, 79, 203, 254, 271–72 pa, 70, 76, 254, 278 ip ad, 94n, 98–99 ppp, 64, 69, 76, 155, 193, 251, 311 ip ad, 166–68, 222–23, 285 ppi, 181 phloat, phloa t, 232 2 32 pimopau,, 60–62, 151 pimopau ptiid, 121, 280 atioxidat ad, 73–74 otio tw autoim autoimmu mu dia ad, 243–46 dtox ad, 273, 276 glutathio ad, 249–50 glutathio 24 9–50 gut ad, 180, 245 lupu ad, ad, 302, 305 toxiity ad, 19, 229–30, 233, 236–38, 243–46, 249, 262–63, 272, 275 pto, 160 pH, 181–84, 207 phytoutit, phyto utit, 43, 54, 100, 161, 163 pi ut, 222–23 pituitay, 108, 109–10 plati, 230, 232, 237–38, 243–44, 249, 262, 272–73 plum, 76, 79, 185, 207, 271–72 PMs, 110, 245–46 polyomiatd diphyl th, 232 polyhloiatd polyhloia td iph iphyl yl (Pcb), 230, 237–38, 244, 288 polyttauoothyl, polyttauo othyl, 232 potato, 64, 193, 272, 311 ip ad, 164, 222 whit, 74, 78–79, 164, 270 poulty,, 76–77, 236 poulty dtox ad, 251, 273 pioti: guid to, 331 gut ad, 203–4, 211 pdio, x, 21, 122, 306 pgay, 21–22, 130, 234, 238, 241, 245 pvtiv pv tiv mdii, 2, 5–6, 22–23 poioti, poio ti, xi, 165, 217 guid to, 331 gut ad, 177–78, 184, 187, 192–93, 201, 203–4, 206, 210–11, 214, 268–69, 308
365
pogto, 60, 114, 247, 304–5 pogto, potagladi, 55 pota, pota , 197, 207–8 2 07–8 poti, 4, 11, 19, 88, 175, 233, 268, 331–33 dtox ad, ad, 248, 251–52, 270, 273, 276–78 ad atig o immu halth, 74, 75 ood ad, 40, 43–46, 64, 66–67, 203, 277–78 glut ad, ad, 15, 45–47, 62, 65, 180, 294 gut ad, 180–82, 187, 197, 203, 208, 210 lupu ad, 302–303 rA ad, 30–31, 193, 308–9 ip ad, 93–97, 101, 158, 160, 162, 165, 169, 220, 280, 285, 287, 289 t ad, 113, 158 Proteus mirabilis, 310 poto pump ihiito (PPI), 44, 182, 184, 186, 199, 206, 208 puddig, 170–71, 207, 224 pu, 8, 14, 21, 45, 120, 231 gut ad, 186, 191 pumpki, pumpki , pumpki d, 55, 58, 76, 82, 94n, 94 n, 154, 207, 271, 276 puttaa putta a au, 168–69 quti, 39 quioa, 45–46, 65, 68–69, 89, 270 ad atig o immu halth, 77, 79–80 ip ad, 95–99, 101–2, 158, 162, 283 ah, 48, 70, 265, 274 lupu ad, ad, 29, 33, 303 ip: gut ad, 215–25 liv ad, 280–90 t ad, 158–71 ad uig ood a mdii, 91–102 ux, 15, 154, 278 ood ad, 45, 63 GerD ad, 199, 207 gut ad, 180–85, 196–97, 199, 207 laxig, 5, 150 guidd, 125–26 o lly athig ad, 145–46 t ad, ad, 106, 113, 121, 123, 125, 132, 136–37, 143–48 tool o, 326–27 vatol, 42, 54
366
idx
humatoid athiti (rA), ix, 2, 7, 13, humatoid 20–24, 48, 129, 242, 256, 293, 296, 308–14 diago o, 51, 191–92 ebV ad, 310–11, 313 ood ad, 37, 45, 53–56, 311 gut ad, 183, 185, 309–11 t ad, 120, 122 upplmt ad, 86, 311 ymptom ympto m o, 23, 30, 33, 37, 55–56, 120, 191, 308–9, 311–12 tt o, o, 30–31, 191, 193, 302, 308–11 toxi ad, ad, 20, 244–46 tatmt tatm t o, o, 53–56, 191–92, 311 tigg ad au o, 16, 309–11 hodiola, 155 i, 45–46, 210, 278 ow, 77, 79–80, 89–90, 97, 270 dtox ad, 270, 276 ad atig o immu halth, 74–80 ood itiviti ad, 65–66, 68–69 ip ad, 97–98, 164, 287 omay,, 80, 276, omay 276 , 285 y, 45, 65, 68, 79, 203, 270 Saccharomyces boulardii, 204, 211, 214, 331 alad, see rip Idx alivay glad, 31, 183, 312–13 aliva tt, 61, 156, 179, 305–6 alivatio, 84, 107, 179 almo, 155, 233 at ad, 55, 99 ip ad, 99–100, 158, 288 almolla, 176 lodma, 2, 22, 33, 240 aood, 4, 76–77, 233–34 see also h a vgtal, 90, 223, 285 d, 15, 46, 58, 66–67, 90, 154, 157, 204, 207, 254, 305, 313 dtox ad, 271, 276–77 ad atig o immu halth, 76–77, 81–82 at ad, 53, 55, 86, 93 Ms ad, 300–301 ip ad, 93–96, 102, 286 see also ut lium, 17, 54, 77, 259, 277 autoimmu thyoid dia ad, 58, 318–19 ip ad, 286, 288 upplmt ad, 84, 86, 329
am d, am oil, 58, 66, 82, 90, 154, 163, 271 x, xuality, xuality, ix, 60, 110, 118, 138, 150, 238, 241, 243–44 homo ad, see tog; ttoto hiitak muhoom, 155 hot-hai atty atty aid, 179–80, 210 id dih, 91 ad atig o immu halth, 74–77 see also rip Idx iu, 12, 154, 186, 196, 265 ood itiviti ad, ad, 64, 66, 71 sjög’ ydom ad, 31, 312 sjög’ ydom, 2, 7, 13, 22, 24, 120, 293, 296, 312–14 ebV ad, 313–14 gut ad, 183–85 ymptom ympto m o, 31–33, 183–84, 309, 312–13 tt o, o, 31, 193, 308–9, 312, 314 tatmtt o, 156, 312–13 tatm ki, ix–xi, 3, 13, 48, 56, 62, 70, 119, 154, 178, 198 lia dia ad, 32–33 lupu ad, ad, 33, 301 rA ad, 30, 33, 308 sjög’ ydom ad, 31, 312 toxi ad, ad, 229, 234, 265 vitiligo ad, 14, 24, 33, 129 lp, 6, 8, 126, 146, 273, 314 cD o, 150–51 glut ad, 50, 61 Gav’ dia ad, 26, 32, 120, 241, 315 gut ad, 192–94, 214 mdiatio ad, ad, 21, 150–51 pimopau ad, 60, 151 t ad, ad, 17, 105, 110, 121, 132, 136–38, 142, 145, 148–51, 268 upplmt o, 150–51, 330 tip o tt, 149–50 toxi ad, ad, 151, 235, 246, 262, 264 lippy lm, 206–7 mall ittial atial ovgo ovgowth wth (sIbO), 185 mokig, 124, 229, 263, 302 moothi, 4, 52, 74, 202 dtox ad, 278–79 ip ad, 217, 282–83 ak, 42, 64, 69, 92, 154, 280 dtox ad, 272–73, 278 ad atig o immu halth, 74, 79–81
idx t ad, 136, 152 see also rip Idx oa oodl, 96–97 oda, 40, 52, 154, 202, 207 ad atig o immu halth, 78–79, 81 olvt,, 229, 249–50, 263, 275 olvt omati vou ytm, 106 o thoat, 27, 314, 317 oup, 282–84 oy,, 15, 90, 154, 268–70, 305 oy dtox ad, ad, 270, 274, 276 ad atig o immu halth, 72, 79 limiatio om dit o, 64–65, 67–68, 184, 189, 193–94, 270, 276 gut ad, 184, 189, 193–94 ip ad, 91, 158, 160n, 160n, 165 itodutio itod utio o, 67, 70–71 itivity to, to, 63–65, 67–68, 70–71 oy au, 64–65, 68, 90, 270 plt, 68, 79, 270 glut ad, ad, 15, 45, 65, 180 pi, 80 dtox ad, 271, 276 ip ad, 162, 166–67, 219 piah, 76, 155, 210, 251, 260 ip ad, 95, 100–101, 161, 221–23, 282–84 pi, pial od, 106, 153 Ms ad, 14, 29–30, 257, 297–98 piituality piituali ty,, 3, 5, 125–26, 128, 327–28 tah, 74, 75, 79, 270 toid, x–xi, 21, 191–92, 275 tvia, 79, 81, 271 ti, 8, 21, 45, 114, 120, 188–89, 191 rA ad, 30, 33, 308 tomah, 107 aid i, 181–86, 197–99, 205–9, 213 t ad, 105–6, 110 see also gut; hatu; ux tool: lood i, xi, 34 dtox ad, ad, 248, 250, 253, 276 gut ad, 192, 195, 197–98 tool tt, 7, 61, 179, 258, 300, 311 gut ad, 184, 186, 189, 205, 209, 212 t, ix–x, 2, 6–9, 105–71, 178–79, 258, 293, 296, 313 aut, 109, 111–14 adal atigu ad, ad, 17–18, 113–14, 119–21, 132–33, 137–42
367
adal glad ad, ad, 106–10, 112–14, 119–23, 129–30, 132–33, 137–43, 148–49, 152, 158, 330 ad autoimmu dia tigg ad au, 14, 17–18, 21 hoi, 17, 111–14, 119–22, 125, 158, 186, 188, 196, 247 xpi o, 109–11, 132 ght-o-ight ght-o -ight i, 106–11, 113, 118–19 ood ad, 105, 110, 113–14, 121, 124–25, 132, 136–38, 141–43, 152, 158, 268–69 gut ad, 105–6, 110, 124–25, 179, 181–83, 185–86, 188, 196, 200 h o, o, 141, 330 homo ad, 14, 17–18, 22, 106–23, 125, 129–30, 137, 140 itio ad, ad, 115, 118–19, 121, 123–24 laky gut ad, 124, 200 mid-ody thiqu ad, 125–28, 132, 145, 148 vou ytm ad, 106–9, 114, 116, 118–19, 125, 129–30, 145, 148, 158 ip ad, 158–71 l-amt o, 130–43, 148 l-a o, o, 132, 135–36, 139–43, 148–53 to ad, ad, 105, 111, 116–19, 127, 130–35, 139–44, 155, 158 t po po ad, 105–14, 116–19, 125, 129 tool o takig hag o, 143–44 toxi ad, ad, 121, 247, 262 tatmt o, 142–57, 330 uga, 66, 83, 88 dtox limiatio dit ad, 269–73 ad atig o immu halth, 72, 74–81 limiatio om om dit o, 52, 59, 74–81, 154, 202, 259, 268, 270–71, 273 gut ad, 202–3, 259 iammatio iammatio ad, 39, 41–42 ip ad, 93–94, 102, 158, 164, 165n, 165n, 280 t ad, ad, 110, 113, 121, 158 uga withdawal, 78 uloapha, uloa pha, 301, 306 ulu,, 250–52, 282–83 ulu uow d, uow oil, 55, 76–77, 82, 271 uhi, 56
368
idx
upplmt, x–xi, 62, 82, 84–88, 121, upplmt, 306–7, 313 adal glad ad, ad, 143, 155–57, 330 dtox ad, ad, 250–53, 260–62, 269, 271, 274, 276–78, 330, 332–33 DHeA ad, 122–23, 330 at ad, 53–56, 84–86, 271 guid to, 329–33 gut ad, 177, 184–85, 187, 199, 201, 203–9, 211, 213–14, 259, 331–32 liv ad, 20, 231, 332–33 Ms ad, 300–301 rA ad, 86, 311 o lp, 150–51, 330 t ad, ad, 122–23, 141–42, 330 vitami ad, ad, 56–58, 84–87, 259, 277, 307, 329 watig, 50, 110, 149, 265 wt, 79, 81, 90, 93, 271 see also uga wt potato, 69, 76, 155 ip ad, 164–65 wllig, 8, 13–14, 38, 45, 265 autoimmu auto immu dia tigg ad, 16, 21 ood itiviti ad, 67, 71 gut ad, 188–89, 191, 193–94, 197, 294 Ms ad, 30, 297 rA ad, 30, 33, 120, 308 t ad, 114, 120 swi had, 55, 76, 155, 161, 260, 276 wodh, 4, 20, 233–34, 258, 263, 319 ympathti vou ytm, 106–9, 107, 114, 118, 130, 145 ytmi lupu ythmatou ythmatou, , see lupu tahii, 58, 69, 90, 97–98, 166–67, 286 tamai, 68, 90, 163, 270 ll, 52, 57–58, 117, 122, 152 gut ad, 176–77, 190 rA ad, 55, 310 t ad, 106, 115 ta, ta dut, 31, 107, 183, 312 ta, 154 dtox ad, 271, 276 g, 54, 59, 77, 87, 150, 301 tlviio, 149–50 tmph, 68, 101, 165, 270 ttil, 108, 109, 122 ttoto, ttot o, 118, 258 adal atigu ad, ad, 17, 157 DHeA ad, 113, 122–23, 157 lupu ad, 304–5
sjög’ ydom ad, 312–13 t ad, 113–14, 122–23 thai, 150 hlp ll, 12, 115–19, 117 gut ad, 179, 189, 190 thoat, 13, 27, 33, 199, 313–14, 317 thym, 80, 204 thymu, 18, 108, 118 thyoid, 1, 22, 27, 50, 58, 86, 108, 109, 121, 194, 258–61, 308 lia dia ad, 25, 306 ood itiviti ad, 67–68 hypothyoidim hypothy oidim ad, ix, 3–4, 28, 314, 316–18 itio ad, 294–95 pimopau ad, 60–62 ip ad, 170, 285–86 toxi ad, ad, 240–41, 318 see also autoimmu thyoid dia; Gav’ dia; dia ; Hahimoto’ thyoiditi thyoid-timulatig homo (sH), 26–28, 61, 316–18 thyoid-timulatig thyoid- timulatig immuoglouli immuoglouli (sI), 26–27, 61, 316 thyoxi (4), 26–28, 61, 315–18 thyoxi tiglig: lia dia ad, 25, 34, 306 dtox ad, 259–61 Ms ad, 29, 34, 257–58, 297 toxi ad, ad, 231, 235, 245 lymphoyt kill ll (ytotoxi ll), 11–13, 23, 57, 88, 115–20, 117, 152 gut ad, 18–19, 176, 179, 189–90 halthy immu ytm ad, 11–12 t ad, 116–20, 123 toophol, toophol , 86 86 tou, 68, 96–97, 165, 270 tomato, 64, 69, 76, 98–99, 155, 193–94, 207, 254, 311 togu, 13, 41, 45, 265 toxi load, 14, 21, 85, 121, 229–31, 244, 256, 279 ood ad, 276–77 liv ad, 230, 239 l-amt o, 262–64, 266–68 toxi, toxiity toxiity,, ix–x, 8–9, 12, 17, 73, 178–80, 229–56, 258–69, 318–19 otio tw autoim autoimmu mu dia ad, 14, 19–21, 233, 236, 239–47, 249–50, 254
idx viomt ad, ix, 6, 19–20, 121, viomt 229–30, 232–33, 236–40, 243–44, 246–47, 249–50, 252, 256, 262–63, 272–73, 279, 293, 295–96 tog ad, 20, 230, 237, 243–46, 290, 306 h ad, 4, 20, 121, 233–34, 238, 244, 254, 258, 263–64, 275, 319 gti ad, 19, 231, 234, 247, 262, 267–68 gut ad, 180, 185–86, 188, 202–3, 230, 245, 259, 268–69, 306 liv ad, 20, 229–31, 239, 246–47, 258, 262, 267, 269, 280 lupu ad, ad, 303–6, 313 duig xpou to, 272–73 lp ad, 151, 235, 246, 262, 264 t ad, ad, 121, 247, 262 ymptom ympto m ad, 231, 235, 238, 264–68 see also havy mtal tauma, 6, 17, 105, 111, 113, 129–34, 147, 188 gulato ll, 12, 57, 59, 116–19 gut ad, 176, 179, 189–90 tiglyid, 202, 210, 301 ip ad, 215, 220 tua, 4, 20, 77, 155, 233–34, 258, 263, 319 tuky,, 69, 155, 243, 258, 270, 276–77 tuky ip ad, 215, 221–22 tyoi, 154–55, 158, 170–71 ul, 29, 33, 225, 303 umohi plum, 185, 207 uiay tat itio, 310 ui, 89 dtox ad, ad, 248, 253, 255, 259–60, 276, 279 toxi ad, ad, 19, 234, 240 vai, 233, 241–43 valia, 150–51 VcA tt, 305 vgtal, 3, 17, 43, 54, 90, 113, 155, 208, 258, 272 uiou, 260, 276, 282, 305 dak g lay, lay, 66, 85, 251, 254, 260, 284, 305 dtox ad, ad, 251, 254, 260, 270–71, 276, 278 ad atig o immu halth, 72–76, 75, 80–82 at ad, 55, 81–82, 86 ood itiviti ad, 68–69
369
gut ad, 202–3, 206–7, 210 lay g, 46, 55, 81–82, 86, 271 liv ad, 231, 238 Ms ad, 300–301 ighthad ad, ad, 64, 193–94, 19 3–94, 311 ip ad, 96–101, 160–68, 220–24, 280, 282–87, 290 poutd, 206–7 upplmt ad, 85–87 vgtaia, vgtaia , 3, 233, 268 dtox ad, 276–77 ip ad, 158, 165 upplmt ad, 86–87, 208 vimia, 303–4 viu, 8–9, 23, 32, 88, 115, 175, 175 , 220, 242, 313–15 amio aid ad, 43–44 gut ad, 181–82, 188, 190, 202, 210 halthy immu ytm ad, 11–12 lik tw autoimmu dia ad, 20–21, 293–96 lupu ad, ad, 302–5, 310, 313 Ms ad, 299–300 rA ad, 310–11, 313 t ad, ad, 118, 121, 123–24 vitami A, 3, 17, 54, 198, 259, 277 ip ad, 286–87, 290 upplmt ad, 57, 84–85 vitami b, 62, 73, 288, 307 adal glad ad, 154–56 dtox ad, ad, 248, 276–77, 279 gut ad, 181–82, 197 ip ad, 96, 158, 161, 163, 165–66, 284, 290 vitami c (aoi (aoi aid), 76, 85 dtox ad, 255–56, 277 ip ad, 163, 285, 290 vitami D, D, 17, 54, 88, 118, 196, 259–60, 286, 304 Ms ad, 259, 300 upplmt upp lmt ad, 56–57, 84, 87, 259, 329 vitami e, 76, 198 dtox ad, 255–56, 277 ip ad, 286, 290 upplmt ad, 85–86, 277, 329 vitami K, 163, 181, 197, 286 vitami, 43, 252, 258 lia dia ad, 47, 307 dtox ad, ad, 248, 255–56, 260, 275–77, 279 ad atig o immu halth, 73, 76–77
370
idx
vitami (cont. (cont.)) gut ad, 181–82, 196–98 ip ad, 96, 100, 158, 160–61, 163, 165–66, 223, 280, 282, 284–88, 290 upplmt uppl mt ad, 56–58, 84–87, 259, 277, 307, 329 vitiligo, 2, 14, 24, 183 t ad, 129–30 ymptom ympto m o, 33, 129 walkig, xi, 34, 36, 145, 153, 235, 273 walut, 55, 82, 213, 271 wat, x, 79, 207 dtox limiatio dit ad, 271, 273, 278 ad, 253–54 toxi ad, ad, 19, 230, 236, 238–39, 243, 246, 266, 272, 275 wight gai, 3, 8, 21, 40, 60, 78, 129, 202, 241, 314 ood ad, 38, 42 gut ad, 191–92 Hahimoto’ thyoiditi ad, 34, 317 toxi ad, 231, 266 wight lo, xi, 18, 27, 38, 40–41, 59, 314 Gav’ dia ad, 26, 34, 120, 241, 315 gut ad, 193–94 rA ad, 30, 308 what, 64, 77, 270 glut ad, ad, 15, 45, 47, 49, 65, 68, 180 pod ood ad, 46–47 ip ad, 93, 96, 102 why,, 66, 68, 94 why 94n, n, 203, 270, 276, 330 whit ou, 16, 52, 113, 280 ad atig o immu halth, 77–80 limiatio om om dit o, 154, 202, 268, 270, 272
wom, 7, 22, 29, 108 DHeA ad, 112–13, 122–23, 157 lupu ad, ad, 28, 301, 304 sjög’ ydom ad, 31, 312–13 toxi ad, ad, 241, 243–46 wok, 64, 81, 263, 273 mid-ody thiqu ad, 127–28 t ad, ad, 105, 116–18, 127–28, 130, 132–33, 135, 144–45 wit, 30, 191, 308 xotog, xotog , 230, 243–47, 305 yat, xi, 44, 115, 124, 175, 300 gut ad, 182, 184–89, 192–93, 195–96, 199, 202–3, 210, 213, 259–60, 269, 294 toxi ad, ad, 230, 259 yoga, 3, 136, 147–48, 153, 273 yogut, 52, 165, 217 adal glad ad, 154–55 dtox ad, ad, 251, 270, 272 ad atig o immu halth, 79–80 ood itiviti ad, 66, 68–69 gut ad, 177, 202–3, 207 zi, 17, 54, 89, 259 dtox ad, ad, 249, 252, 256, 277 gut ad, 182, 211 ip ad, 166–67, 286 ou o, 76, 87 upplmt ad, 58, 84, 87, 329 zouli, 187–88
